M
Marie-Claire Arrieta
Researcher at University of Calgary
Publications - 69
Citations - 5700
Marie-Claire Arrieta is an academic researcher from University of Calgary. The author has contributed to research in topics: Microbiome & Medicine. The author has an hindex of 23, co-authored 54 publications receiving 4294 citations. Previous affiliations of Marie-Claire Arrieta include University of Alberta & University of British Columbia.
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Journal ArticleDOI
Early infancy microbial and metabolic alterations affect risk of childhood asthma
Marie-Claire Arrieta,Leah T. Stiemsma,Pedro A. Dimitriu,Lisa Thorson,Shannon L. Russell,Sophie Yurist-Doutsch,Boris Kuzeljevic,Matthew J. Gold,Heidi Britton,Diana L. Lefebvre,Padmaja Subbarao,Piush J. Mandhane,Allan B. Becker,Kelly M. McNagny,Malcolm R. Sears,Tobias R. Kollmann,William W. Mohn,Stuart E. Turvey,B. Brett Finlay +18 more
TL;DR: It is reported in a longitudinal human study that infants at risk of asthma have transient gut microbial dysbiosis during the first 100 days of life, and certain bacterial genera were decreased in these children, suggesting a potential causative role of the loss of these microbes.
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A critical assessment of the “sterile womb” and “in utero colonization” hypotheses: implications for research on the pioneer infant microbiome
TL;DR: Current scientific evidence does not support the existence of microbiomes within the healthy fetal milieu, which has implications for the development of clinical practices that prevent microbiome perturbations after birth and the establishment of future research priorities.
Journal ArticleDOI
The Intestinal Microbiome in Early Life: Health and Disease
TL;DR: A greater understanding of how the early-life gut microbiota impacts the authors' immune development could potentially lead to novel microbial-derived therapies that target disease prevention at an early age.
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Alterations in intestinal permeability
TL;DR: This review describes barrier function of the intestine, the structure of the tight junction, methods to evaluate intestinal permeability, and most importantly the relevance of abnormal permeability to disease.
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Reducing small intestinal permeability attenuates colitis in the IL10 gene-deficient mouse
TL;DR: It is suggested that increased intestinal permeability may be an important aetiological event in the development of colitis in IL10−/− mice and by doing so colonic disease is prevented or attenuated.