M
Mario F. Mendez
Researcher at University of California, Los Angeles
Publications - 383
Citations - 22480
Mario F. Mendez is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Frontotemporal dementia & Dementia. The author has an hindex of 65, co-authored 351 publications receiving 18961 citations. Previous affiliations of Mario F. Mendez include Ronald Reagan UCLA Medical Center & Western University of Health Sciences.
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Journal ArticleDOI
Brain and ventricular volumetric changes in frontotemporal lobar degeneration over 1 year
D. S. Knopman,C. R. Jack,Joel H. Kramer,B. F. Boeve,Richard J. Caselli,Neil Graff-Radford,Mario F. Mendez,Bruce L. Miller,Nathaniel D. Mercaldo +8 more
TL;DR: Multicenter, serial measurements of whole brain volume (WBV) and ventricular volume (VV) from magnetic resonance scans were feasible in patients with frontotemporal lobar degeneration (FTLD) and were correlated with decline on clinical global and cognitive measures.
Journal Article
The unique predisposition to criminal violations in frontotemporal dementia.
TL;DR: It is indicated that those with right temporal FTD retain the capacity to tell right from wrong but have the slow and insidious loss of the capacity for moral rationality.
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Fluent versus nonfluent primary progressive aphasia: a comparison of clinical and functional neuroimaging features.
TL;DR: Fluent and nonfluent forms of PPA are clinically distinguishable by letter fluency, single word comprehension, object naming, and types of paraphasic errors, and there is a large amount of overlap between dysfunctional anatomic regions associated with these syndromes.
Journal ArticleDOI
Mild traumatic brain injury from primary blast vs. blunt forces: post-concussion consequences and functional neuroimaging.
Mario F. Mendez,Emily M. Owens,Gholam R. Berenji,Dominique Peppers,Li-Jung Liang,Eliot A. Licht +5 more
TL;DR: This pilot study suggests that pure blast force mTBI may have greater post-concussive sequelae including deficits in attentional control and regional brain metabolism, compared to blunt mT BI.
Journal ArticleDOI
Neuropathology of Autosomal Dominant Alzheimer Disease in the National Alzheimer Coordinating Center Database
John M. Ringman,John M. Ringman,Sarah E. Monsell,Denise W. Ng,Yan Zhou,Andy Nguyen,Giovanni Coppola,Victoria Van Berlo,Mario F. Mendez,Spencer Tung,Sandra Weintraub,Marek-Marsel Mesulam,Eileen H. Bigio,Darren R. Gitelman,Darren R. Gitelman,Darren R. Gitelman,Amanda Fisher-Hubbard,Roger L. Albin,Harry V. Vinters +18 more
TL;DR: The finding of more severe neurofibrillary pathology and CAA in ADAD, particularly in carriers of PSEN1 mutations beyond codon 200, warrants consideration when designing trials to treat or prevent ADAD.