M
Marise B. Parent
Researcher at Georgia State University
Publications - 73
Citations - 2355
Marise B. Parent is an academic researcher from Georgia State University. The author has contributed to research in topics: Muscimol & Hippocampus. The author has an hindex of 27, co-authored 69 publications receiving 2211 citations. Previous affiliations of Marise B. Parent include University of California, Irvine & University of Alberta.
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Journal ArticleDOI
Septohippocampal acetylcholine: involved in but not necessary for learning and memory?
Marise B. Parent,Mark G. Baxter +1 more
TL;DR: It is suggested that although hippocampal ACh is involved in memory in the intact brain, it is not necessary for many aspects of hippocampal memory function.
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Neuromodulatory systems and memory storage: Role of the amygdala
James L. McGaugh,Ines B. Introini-Collison,Larry Cahill,Claudio Castellano,Carla Dalmaz,Carla Dalmaz,Marise B. Parent,Cedric L. Williams +7 more
TL;DR: Findings suggest that the amygdala integrates the memory-modulating effects of neuromodulatory systems activated by learning experiences, and appears to involve activation of a cholinergic system.
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A high fructose diet impairs spatial memory in male rats.
TL;DR: The impaired spatial water maze retention performance seen with the high fructose diet may have been attributable, at least in part, to fructose-induced increases in plasma triglycerides.
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Posttraining infusion of lidocaine into the amygdala basolateral complex impairs retention of inhibitory avoidance training.
TL;DR: The time- and footshock-dependent retrograde impairment of memory produced by posttraining reversible inactivation of the basolateral complex suggests that this region of the amygdala is involved in the consolidation of memory for inhibitory avoidance training.
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Increased training in an aversively motivated task attenuates the memory-impairing effects of posttraining N-methyl-D-aspartate-induced amygdala lesions.
TL;DR: The finding that AC-lesioned animals displayed greater locomotor activity on the retention test compared with nonlesioned controls suggests that the increased activity may have contributed to the impaired inhibitory avoidance retention performance.