M
Mark J. Edwards
Researcher at St George's, University of London
Publications - 398
Citations - 20432
Mark J. Edwards is an academic researcher from St George's, University of London. The author has contributed to research in topics: Dystonia & Psychogenic disease. The author has an hindex of 63, co-authored 365 publications receiving 16293 citations. Previous affiliations of Mark J. Edwards include St George’s University Hospitals NHS Foundation Trust & University College London.
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Clinical applications of transcranial magnetic stimulation in patients with movement disorders
TL;DR: The theoretical background to TMS techniques are described, the difficulties in bringing the technique into regular clinical diagnostic practice will be discussed, and the evidence for the potential of repetitive TMS as a therapeutic tool in patients with movement disorders will be reviewed.
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The cerebellum in dystonia - help or hindrance?
TL;DR: It is proposed that abnormal, likely compensatory activity of the cerebellum is an important factor within pathophysiological models of dystonia.
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Basal ganglia, dopamine and temporal processing: performance on three timing tasks on and off medication in Parkinson's disease
TL;DR: The data suggest that the integrity of the basal ganglia is necessary for 'typical' time production in the seconds range as well as for time reproduction at shorter intervals and that the time production task uses neural mechanisms distinct from those used in the other two timing tasks.
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Neurobiology of functional (psychogenic) movement disorders
TL;DR: New understanding of the neurobiology of FMD forms an important part of reappraising the way that patients with FMD (and other functional disorders) are characterized and treated and provides a testable framework for further exploring the pathophysiology of these common causes of ill health.
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Environmental factors in Tourette syndrome
TL;DR: Recent data point to intrinsically altered immune regulation in TS, which might predispose to both infections and autoimmune mechanisms; however, evidence of cell-mediated and antibody-mediated autoimmunity in TS is still insufficient.