M
Martin L. Adamo
Researcher at University of Texas Health Science Center at San Antonio
Publications - 107
Citations - 5871
Martin L. Adamo is an academic researcher from University of Texas Health Science Center at San Antonio. The author has contributed to research in topics: Insulin-like growth factor & Insulin. The author has an hindex of 44, co-authored 107 publications receiving 5650 citations. Previous affiliations of Martin L. Adamo include National Institutes of Health & Pennsylvania State University.
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Journal ArticleDOI
Deletion of the G Protein-Coupled Receptor 30 Impairs Glucose Tolerance, Reduces Bone Growth, Increases Blood Pressure, and Eliminates Estradiol-Stimulated Insulin Release in Female Mice
Ulrika E.A. Mårtensson,S Albert Salehi,Sara H Windahl,Maria F. Gomez,Karl Swärd,Joanna Daszkiewicz-Nilsson,Anna Wendt,Niklas Andersson,Per Hellstrand,Per-Olof Grände,Christer Owman,Clifford J. Rosen,Martin L. Adamo,Ingmar Lundquist,Patrik Rorsman,Bengt-Olof Nilsson,Claes Ohlsson,Björn Olde,L. M. Fredrik Leeb-Lundberg +18 more
TL;DR: The findings show that GPR30 is important for several metabolic functions in female mice, including estradiol-stimulated insulin release, which is associated with decreased insulin expression and release in vivo and in vitro in isolated pancreatic islets.
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Developmental regulation of the rat insulin-like growth factor I receptor gene
Haim Werner,Michael Woloschak,Martin L. Adamo,Zila Shen-Orr,Charles T. Roberts,Derek LeRoith +5 more
TL;DR: Overall, steady-state IGF-I receptor mRNA levels decreased dramatically during postnatal development; however, the extent of the decrease differed among the various tissues studied.
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Regulation by fasting of rat insulin-like growth factor I and its receptor. Effects on gene expression and binding.
TL;DR: The data demonstrate that the change in IGF-I and IGF/I receptor mRNA levels during fasting is quantitatively different in different tissues and suggest that regulation of IGF-i and IGF- I receptor gene expression by fasting is discoordinate.
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Insulin and insulin-like growth factor receptors in the nervous system.
TL;DR: Support is added to the suggestion that insulin and IGFs are important neuroactive substances, regulating growth, development, and metabolism in the brain, with the highest levels appearing in fetal or perinatal life.
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Minireview: Mechano-growth factor: a putative product of IGF-I gene expression involved in tissue repair and regeneration.
TL;DR: The relationship of the Igf1 gene to MGF is discussed and actions of synthetic MGF from any known product of Igf 1 are differentiated and the role of MGF in satellite cell activation, aging, neuroprotection, and signaling will be discussed.