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L. M. Fredrik Leeb-Lundberg

Researcher at Lund University

Publications -  58
Citations -  4610

L. M. Fredrik Leeb-Lundberg is an academic researcher from Lund University. The author has contributed to research in topics: Receptor & Kinin. The author has an hindex of 31, co-authored 57 publications receiving 4303 citations. Previous affiliations of L. M. Fredrik Leeb-Lundberg include Howard Hughes Medical Institute & University of Texas System.

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International Union of Pharmacology. XLV. Classification of the Kinin Receptor Family: from Molecular Mechanisms to Pathophysiological Consequences

TL;DR: This review is a comprehensive presentation of the current understanding of B1 and B2 receptors in terms of molecular and cell biology, physiology, pharmacology, and involvement in human disease and drug development.
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Amphetamine-induced loss of human dopamine transporter activity: an internalization-dependent and cocaine-sensitive mechanism.

TL;DR: Evidence is presented that DAT substrates, including AMPH and DA, cause internalization of human DAT, thereby reducing transport capacity, and acute application of AMPH would reduce DA uptake not only by direct competition for uptake, but also by reducing the available cell-surface DAT.
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Bradykinin Sequesters B2 Bradykinin Receptors and the Receptor-coupled Gα Subunits Gαq and Gαiin Caveolae in DDT1 MF-2 Smooth Muscle Cells

TL;DR: It is shown that the vasoactive peptide agonist bradykinin when bound to B2 BK receptors on DDT1 MF-2 smooth muscle cells promotes the recruitment and sequestration of the occupied receptors and the receptor-coupled G-protein α subunits Gαq and Gαi in caveolae.
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Lysophosphatidic Acid Binds to and Activates GPR92, a G Protein-Coupled Receptor Highly Expressed in Gastrointestinal Lymphocytes

TL;DR: The ligand binding, activation, and tissue distribution of the orphan G protein-coupled receptor (GPCR) GPR92 were studied and it was found that it is the most abundant GPCR activated by LPA found in the small intestinal intraepithelial CD8+ cytotoxic T cells.