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Mary A. Cutting

Researcher at Walter Reed Army Institute of Research

Publications -  9
Citations -  283

Mary A. Cutting is an academic researcher from Walter Reed Army Institute of Research. The author has contributed to research in topics: Alpha (ethology) & Platelet. The author has an hindex of 5, co-authored 9 publications receiving 276 citations.

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Mechanisms of hemorrhage in dengue without circulatory collapse.

TL;DR: The data suggest that vascular alteration may be the principal factor involved in the association of thrombocytopenia and hemorrhage with disease severity, and that hemorrhage in dengue without circulatory collapse is most likely due to activation of platelets rather than coagulopathy, which is well compensated.
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Platelet adhesion to dengue-2 virus-infected endothelial cells.

TL;DR: It is demonstrated that human platelets adhere to dengue-2 virus-stimulated HUVEC and this interaction could contribute to the thrombocytopenia observed during infection.
Journal Article

Effects of anti-CD33 blocked ricin immunotoxin on the capacity of CD34+ human marrow cells to establish in vitro hematopoiesis in long-term marrow cultures.

TL;DR: Findings support the potential usefulness of anti-CD33-bR for in vitro marrow purging or in vivo treatment to eliminate CD33+ leukemic clones, while sparing normal CD34+/CD34- stem cells that support normal hematopoiesis and hematoietic reconstitution in vivo.
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Cardiovascular toxicity of human cross-linked hemoglobin in a rabbit endotoxemia model.

TL;DR: In an animal model of nonlethal endotoxemia, infusion of alpha alpha Hb significantly increases mortality and the data suggest that mortality may be due to the acute increased cardiopulmonary toxicity of alphaalpha Hb in animals with underlying endotoxinemia.
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Assessment of ω-fatty-acid-supplemented human platelets for potential improvement in long-term storage

TL;DR: The ω−3-based agents protect the platelets from damage during the washing procedure as demonstrated in vitro by improved platelet resuspension, low %RPC, high stimulus-responsive ATP secretion and a reduction in blood loss in vivo.