M
Massimo Collino
Researcher at University of Turin
Publications - 143
Citations - 5071
Massimo Collino is an academic researcher from University of Turin. The author has contributed to research in topics: Inflammasome & Inflammation. The author has an hindex of 37, co-authored 131 publications receiving 4139 citations.
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Journal ArticleDOI
Modulation of the oxidative stress and inflammatory response by PPAR-γ agonists in the hippocampus of rats exposed to cerebral ischemia/reperfusion
Massimo Collino,Manuela Aragno,Raffaella Mastrocola,Margherita Gallicchio,Arianna Carolina Rosa,Chiara Dianzani,Oliviero Danni,C. Thiemermann,Roberto Fantozzi +8 more
TL;DR: Significant evidence is provided that PPAR-gamma agonists exert protective effects in a rat model of mild forebrain ischemia/reperfusion injury by inhibiting oxidative stress and excessive inflammatory response by inhibition of COX-2 protein expression and activation of MAPKs and NF-kappaB.
Journal ArticleDOI
Anti-apoptotic and anti-inflammatory effects of hydrogen sulfide in a rat model of regional myocardial i/r
Ahila Sivarajah,Massimo Collino,Mohammed Yasin,Elisa Benetti,Margherita Gallicchio,Emanuela Mazzon,Salvatore Cuzzocrea,Roberto Fantozzi,Christoph Thiemermann +8 more
TL;DR: It is demonstrated that the cardioprotective effect of NaHS is secondary to a combination of antiapoptotic and anti-inflammatory effects.
Journal ArticleDOI
Generation of endogenous hydrogen sulfide by cystathionine γ -lyase limits renal ischemia/reperfusion injury and dysfunction
Pinpat Tripatara,Pinpat Tripatara,Nimesh S. A. Patel,Massimo Collino,Margherita Gallicchio,Julius Kieswich,Sara Castiglia,Elisa Benetti,Keith N. Stewart,Paul A.J. Brown,Mohammed M. Yaqoob,Roberto Fantozzi,Christoph Thiemermann +12 more
TL;DR: The findings suggest that the synthesis of endogenous hydrogen sulfide by CSE is essential to protect the kidney against ischemia/reperfusion injury and dysfunction and aids in the recovery of renal function following ischemIA/rePerfusion.
Journal ArticleDOI
Oxidative stress and inflammatory response evoked by transient cerebral ischemia/reperfusion: effects of the PPAR-alpha agonist WY14643.
Massimo Collino,Manuela Aragno,Raffaella Mastrocola,Elisa Benetti,Margherita Gallicchio,Chiara Dianzani,Oliviero Danni,Christoph Thiemermann,Roberto Fantozzi +8 more
TL;DR: It is proposed that WY14643 protects the brain against excessive oxidative stress and inflammation and may thus be useful in treating stroke.
Journal ArticleDOI
PPARgamma stimulation promotes mitochondrial biogenesis and prevents glucose deprivation-induced neuronal cell loss.
Gianluca Miglio,Arianna Carolina Rosa,Lorenza Rattazzi,Massimo Collino,Grazia Lombardi,Roberto Fantozzi +5 more
TL;DR: The data indicate that a prolonged PPARgamma stimulation, by repeated administration of nanomolar pioglitazone or rosiglitaz one concentrations, decreases GD-induced loss of differentiated SH-SY5Y cells, and suggest that mitochondrial biogenesis may contribute to these effects.