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Matthew Ellis

Researcher at University of Southampton

Publications -  53
Citations -  1706

Matthew Ellis is an academic researcher from University of Southampton. The author has contributed to research in topics: Duchenne muscular dystrophy & Neonatal encephalopathy. The author has an hindex of 18, co-authored 48 publications receiving 1293 citations. Previous affiliations of Matthew Ellis include UCL Institute of Neurology & Queen's University.

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Evidence for human transmission of amyloid-β pathology and cerebral amyloid angiopathy

TL;DR: The marked deposition of parenchymal and vascular Aβ in these relatively young patients with iCJD, in contrast with other prion disease patients and population controls, is consistent with iatrogenic transmission of Aβ pathology in addition to CJD and suggests that healthy exposed individuals may also be at risk of i atrogenic Alzheimer’s disease and cerebral amyloid angiopathy.
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Risk factors for neonatal encephalopathy in Kathmandu, Nepal, a developing country: unmatched case-control study

TL;DR: Intrapartum risk factors remain important for neonatal encephalopathy in developing countries and there is some evidence of a protective effect from antenatal care.
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Quantum localization and delocalization of charge carriers in organic semiconducting crystals.

TL;DR: By solving the time-dependent electronic Schrödinger equation coupled to nuclear motion for eight organic molecular crystals, the excess charge carrier forms a polaron delocalized over up to 10–20 molecules in the most conductive crystals.
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Factors affecting home delivery in the Kathmandu Valley, Nepal

TL;DR: It is concluded that poor education and multiparity rather than poverty per se increase the risk of a home delivery in Kathmandu, Nepal and training TBAs in this setting would probably not be cost-effective.
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The ventrolateral medulla and medullary raphe in sudden unexpected death in epilepsy

TL;DR: Changes in pre-Bötzinger region neurons and modulatory medullary neuropeptidergic and monoaminergic systems, including galanin, somatostatin and serotonin are revealed, which may be sequelae of seizures and risk factors for SUDEP through defective respiratory homeostasis.