M
Mattia Albiero
Researcher at University of Padua
Publications - 89
Citations - 5779
Mattia Albiero is an academic researcher from University of Padua. The author has contributed to research in topics: Progenitor cell & Bone marrow. The author has an hindex of 35, co-authored 78 publications receiving 4807 citations.
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Journal ArticleDOI
Angiogenic Abnormalities in Diabetes Mellitus: Mechanistic and Clinical Aspects.
TL;DR: Dissecting the molecular drivers of tissue-specific alterations of angio-/vasculogenesis is an important challenge to devise new therapeutic approaches to normalize angiogenesis in diabetes.
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Diabetes Limits Stem Cell Mobilization Following G-CSF but Not Plerixafor.
Gian Paolo Fadini,Mark A. Fiala,Roberta Cappellari,Marianna D’Anna,Soo Park,Nicol Poncina,Lisa Menegazzo,Mattia Albiero,John F. DiPersio,Keith Stockerl-Goldstein,Angelo Avogaro +10 more
TL;DR: Testing whether the CXCR4 antagonist plerixafor, differently from G-CSF, is effective in mobilizing hematopoietic stem cell (HSC) mobilization in patients with diabetes found it was equally able to mobilize CD34+ HSCs in the two groups, whereas in historical data, G- CSF was less effective in Patients with diabetes.
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A perspective on NETosis in diabetes and cardiometabolic disorders.
Gian Paolo Fadini,Lisa Menegazzo,Valentina Scattolini,M. Gintoli,Mattia Albiero,Angelo Avogaro +5 more
TL;DR: NETosis appears to be part of an abnormal response to damage in diabetes that can promote or aggravate end-organ complications, making it a novel player in the pathobiology of cardiovascular disease.
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NAD(+)-dependent SIRT1 deactivation has a key role on ischemia-reperfusion-induced apoptosis.
Arianna Cattelan,Giulio Ceolotto,Sergio Bova,Mattia Albiero,Maniselvan Kuppusamy,Sara De Martin,Andrea Semplicini,Gian Paolo Fadini,Saula Vigili de Kreutzenberg,Angelo Avogaro +9 more
TL;DR: The interplay among FoxO1, SIRT1, NAD(+), AMPK, HSP, and SIRT6 depicts a complex molecular network that protects the heart from apoptosis during IR and may be susceptible to therapeutic interventions.
Journal ArticleDOI
Procalcific phenotypic drift of circulating progenitor cells in type 2 diabetes with coronary artery disease.
Gian Paolo Fadini,Mattia Albiero,Lisa Menegazzo,Elisa Boscaro,Carlo Agostini,Saula Vigili de Kreutzenberg,Marcello Rattazzi,Angelo Avogaro +7 more
TL;DR: In this paper, the pro-calcific versus endothelial differentiation status of circulating CD34+ progenitor cells was determined by expression of VEGFR-2 (KDR) and Pro-Calcific polarization of circulating progenitors, which may contribute to vascular calcification in these patients.