M
Meijia Zhang
Researcher at University of Minnesota
Publications - 36
Citations - 916
Meijia Zhang is an academic researcher from University of Minnesota. The author has contributed to research in topics: Oocyte & Phosphorylation. The author has an hindex of 17, co-authored 36 publications receiving 815 citations. Previous affiliations of Meijia Zhang include South China University of Technology.
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Journal ArticleDOI
The signal pathway of gonadotrophins-induced mammalian oocyte meiotic resumption.
TL;DR: A detailed appreciation of different FSH and LH-activated signaling pathways in mammalian oocytes will be needed in understanding their actions in follicular development and oocyte maturation.
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Epidermal growth factor receptor activation by protein kinase C is necessary for FSH-induced meiotic resumption in porcine cumulus–oocyte complexes
Xiufen Chen,Bo Zhou,Jun Yan,Baoshan Xu,Ping Tai,Junxia Li,Shiming Peng,Meijia Zhang,Guoliang Xia +8 more
TL;DR: The results supported that endogenously produced EGFR activator(s), possibly AR and EGFR activation, in cumulus cells is necessary for FSH-induced porcine oocyte meiotic resumption, and suggested that EGFRactivation, by PKC signal pathway, participates in F SHS-induced Porcine Oocyte meiosis resumption.
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Immunohistochemical localization of inducible and endothelial nitric oxide synthase in porcine ovaries and effects of NO on antrum formation and oocyte meiotic maturation.
TL;DR: The results demonstrate that porcine ovaries have distinct cell-specific expression of both eNOS and iNOS, and that NO derived from both NOS is actively involved in meiotic resumption and that exogenous NO inhibits the antrum formation.
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MAPK3/1 Participates in the Activation of Primordial Follicles through mTORC1-KITL Signaling.
TL;DR: The results of the present study suggest that MAPK3/1 participates in primordial follicle activation through mTORC1‐KITL signaling, and used phosphatase and tensin homolog deleted on chromosome 10 (PTEN) inhibitor bpV(HOpic) to promotePrimordial follicles activation.
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Ursolic acid prevents endoplasmic reticulum stress-mediated apoptosis induced by heat stress in mouse cardiac myocytes.
Ying Yang,Changwu Li,Xi Xiang,Zhaolai Dai,Jianyu Chang,Ming Zhang,Hong Cai,Hua Zhang,Meijia Zhang,Yuming Guo,Zhenlong Wu +10 more
TL;DR: The striking finding that ursolic acid has both anti-apoptotic and antioxidative activities against ER stress-associated myocardial damage suggests that supplementation of ursoli acid might be a potential strategy to reduce the detrimental effects of heat stress in cardiomyocytes.