M
Menachem Hanani
Researcher at Hebrew University of Jerusalem
Publications - 149
Citations - 5477
Menachem Hanani is an academic researcher from Hebrew University of Jerusalem. The author has contributed to research in topics: Myenteric plexus & Satellite glial cell. The author has an hindex of 37, co-authored 144 publications receiving 4717 citations. Previous affiliations of Menachem Hanani include Woods Hole Oceanographic Institution & Mayo Clinic.
Papers
More filters
Journal ArticleDOI
Satellite glial cells in sensory ganglia: from form to function
TL;DR: This review presents information that has been accumulated recently on the physiology and pharmacology of satellite glial cells in sensory ganglia and proposes that these cells have a role in pathological changes in the ganglia.
Journal ArticleDOI
Coupled Activation of Primary Sensory Neurons Contributes to Chronic Pain
Yu Shin Kim,Yu Shin Kim,Michael Anderson,Kyoungsook Park,Qin Zheng,Amit Agarwal,Catherine Gong,Saijilafu,Saijilafu,Le Anne Young,Shao Qiu He,Pamela Colleen LaVinka,Feng Quan Zhou,Dwight E. Bergles,Menachem Hanani,Yun Guan,David C. Spray,Xinzhong Dong +17 more
TL;DR: An imaging technique was developed that allowed for simultaneously monitor the activities of >1,600 neurons/DRG in live mice and discovered a striking neuronal coupling phenomenon that adjacent neurons tend to activate together following tissue injury, which contributes to pain hypersensitivity by "hijacking" neighboring neurons through gap junctions.
Journal ArticleDOI
Satellite glial cells in sensory ganglia: their possible contribution to inflammatory pain.
Pavel Dublin,Menachem Hanani +1 more
TL;DR: The results show that augmented glial coupling is one of the major events occurring in DRG following inflammation, and provides indirect support for the idea that augmented intercellular coupling might contribute to chronic pain.
Journal ArticleDOI
Glial cell plasticity in sensory ganglia induced by nerve damage.
TL;DR: It is proposed that axotomy induces growth of perineuronal SC sheaths, leading to contacts betweenSCs enveloping adjacent neurons and to formation of new gap junctions between SCs, which may contribute to neuropathic pain.
Journal ArticleDOI
The contribution of satellite glial cells to chemotherapy-induced neuropathic pain.
TL;DR: The hypothesis that satellite glial cells in dorsal root ganglia (DRGs) are altered in chemotherapy‐induced peripheral neuropathy models and contribute to neuropathic pain is tested.