M
Michael O'Dell
Researcher at Norwich Research Park
Publications - 14
Citations - 1202
Michael O'Dell is an academic researcher from Norwich Research Park. The author has contributed to research in topics: Gene & Chalcone synthase. The author has an hindex of 11, co-authored 14 publications receiving 1183 citations.
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Journal ArticleDOI
A molecular description of telomeric heterochromatin in Secale species.
TL;DR: The physical properties, sequence divergence and chromosomal distribution of six different repeated sequences in Secale cereale (cultivated rye) are described and it is suggested that each of the S. cereale-specific repeats may have evolved by the insertion of DNA elements into an array of simple repeats followed by amplification of the portion of the array containing the inserted sequence.
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RNA-Mediated RNA Degradation and Chalcone Synthase A Silencing in Petunia
TL;DR: A model involving cycles of RNA-RNA pairing between complementary sequences followed by endonucleolytic RNA cleavages to describe how RNA degradation is likely to be promoted is presented.
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The genetic control of nucleolus formation in wheat
TL;DR: There is a frequently, but not invariably, seen correlation between rRNA gene number and nucleolus size, however the relative size of theucleolus formed depends principally upon the proportion of the total active rRNA genes in the cell which are localised at the nucleolUS organiser in question.
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Repeated sequence DNA comparisons between Triticum and Aegilops species
TL;DR: The results are consistent with the hypothesis that speciation is accompanied by quantitative changes in the repeated sequence complements of genomes, and using a DNA probe from Aegilops speltoides that contains the most highly repeated sequences, diploid Aegilop species could be distinguished from diploids Triticum species.
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Epigenetic Inactivation of Chalcone Synthase-A Transgene Transcription in Petunia Leads to a Reversion of the Post-Transcriptional Gene Silencing Phenotype
TL;DR: The authors showed that post-transcriptional gene silencing (PTGS) can interfere with the initiation of transgene transcription, leading to a reversion of the PTGS phenotype.