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Michael Pecka

Researcher at Ludwig Maximilian University of Munich

Publications -  35
Citations -  2159

Michael Pecka is an academic researcher from Ludwig Maximilian University of Munich. The author has contributed to research in topics: Sound localization & Sensory system. The author has an hindex of 15, co-authored 33 publications receiving 1906 citations. Previous affiliations of Michael Pecka include University College London & Max Planck Society.

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Mechanisms of Sound Localization in Mammals

TL;DR: Recent evidence concerning the nature of the neural representation of auditory space in the mammalian brain is reviewed and advances in the understanding of mammalian subcortical processing of auditory spatial cues that challenge the "textbook" version of sound localization are elaborate.
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Interaural Time Difference Processing in the Mammalian Medial Superior Olive: The Role of Glycinergic Inhibition

TL;DR: In this paper, the authors present physiological and pharmacological data from in vivo extracellular MSO recordings in anesthetized gerbils, and conclude that, for proper ITD processing, not only is inhibition necessary, but it must also be precisely timed.
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Tuning of Ranvier node and internode properties in myelinated axons to adjust action potential timing

TL;DR: Unexpected structural specializations in the Ranvier nodes and internodes of auditory brainstem axons involved in sound localization are reported, showing individual anatomical parameters of myelinated axons can be tuned to optimize pathways involved in temporal processing.
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The natural history of sound localization in mammals – a story of neuronal inhibition

TL;DR: It is argued that mammalian and avian phylogeny of spatial hearing is characterized by a convergent evolution of hearing air-borne sounds rather than by homology, which will significantly increase the explanatory power of studies of spatial processing in both mammals and birds.
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Synaptic transmission at the calyx of Held under in vivo like activity levels.

TL;DR: It is proposed that some in vivo auditory synapses are in a tonic state of reduced EPSC amplitudes as a consequence of high spontaneous spiking and this in vivo-like conditioning has important consequences for the encoding of signals throughout the auditory pathway.