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Miki Fujimura

Researcher at Tohoku University

Publications -  338
Citations -  13058

Miki Fujimura is an academic researcher from Tohoku University. The author has contributed to research in topics: Moyamoya disease & Cerebral blood flow. The author has an hindex of 50, co-authored 289 publications receiving 11341 citations. Previous affiliations of Miki Fujimura include Stanford University.

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Angiographic circulation time and cerebral blood flow during balloon test occlusion of the internal carotid artery

TL;DR: Testing the hypothesis that the laterality of the hemispheric circulation time (HCT) of the contrast medium at cerebral angiography would reflect bilateral asymmetry of the cerebral blood flow (CBF) during BTO found it strongly correlated with the asymmetry ratio of the CBF.
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Development of a de novo arteriovenous malformation after bilateral revascularization surgery in a child with moyamoya disease.

TL;DR: A 14-year-old girl developed an AVM in the right occipital lobe during the 4-year postoperative period following successful bilateral revascularization surgeries, and this case may provide insight into the dynamic pathology of AVMs.
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Preventive Effect of Clazosentan against Cerebral Vasospasm after Clipping Surgery for Aneurysmal Subarachnoid Hemorrhage in Japanese and Korean Patients

TL;DR: It is suggested that clazosentan prevents cerebral vasospasm and subsequent cerebral infarction, and could thereby improve outcomes after performing a clipping surgery for aSAH in Japanese and Korean patients.
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Endovascular Treatments in Combination with Extracranial-Intracranial Bypass for Complex Intracranial Aneurysms

TL;DR: Combined endov vascular and surgical bypass procedures are useful for the treatment of complex intracranial aneurysms when conventional surgical or endovascular techniques are not feasible and show acceptable rates of morbidity and mortality.
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Increased serum production of soluble CD163 and CXCL5 in patients with moyamoya disease: Involvement of intrinsic immune reaction in its pathogenesis.

TL;DR: The results indicate that patients with MMD may have increased autoimmune activity, and shed light on the pathogenesis of MMD via CD163+ M2-polarized macrophages.