M
Miki Fujimura
Researcher at Tohoku University
Publications - 338
Citations - 13058
Miki Fujimura is an academic researcher from Tohoku University. The author has contributed to research in topics: Moyamoya disease & Cerebral blood flow. The author has an hindex of 50, co-authored 289 publications receiving 11341 citations. Previous affiliations of Miki Fujimura include Stanford University.
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Journal ArticleDOI
Manganese Superoxide Dismutase Affects Cytochrome c Release and Caspase-9 Activation After Transient Focal Cerebral Ischemia in Mice.
TL;DR: The current study revealed Mn-SOD might affect cytochrome c translocation and downstream caspase activation in the mitochondrial-dependent cell death pathway after transient FCI.
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Efficacy of Prophylactic Blood Pressure Lowering according to a Standardized Postoperative Management Protocol to Prevent Symptomatic Cerebral Hyperperfusion after Direct Revascularization Surgery for Moyamoya Disease
TL;DR: Prophylactic blood pressure lowering prevents symptomatic cerebral hyperperfusion after STA-MCA anastomosis in patients with moyamoya disease.
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Angiographic features of hemorrhagic moyamoya disease with high recurrence risk: a supplementary analysis of the Japan Adult Moyamoya Trial
Takeshi Funaki,Jun Takahashi,Kiyohiro Houkin,Satoshi Kuroda,Shigekazu Takeuchi,Miki Fujimura,Yasutake Tomata,Susumu Miyamoto +7 more
TL;DR: Choroidal anastomosis and PCA involvement are characteristic of posterior hemorrhage in moyamoya disease and might be considered a potential source of posterior bleeding at high risk of rebleeding.
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Temporal profile of the vascular anatomy evaluated by 9.4-T magnetic resonance angiography and histopathological analysis in mice lacking RNF213: A susceptibility gene for moyamoya disease
Shinya Sonobe,Miki Fujimura,Kuniyasu Niizuma,Yasuo Nishijima,Akira Ito,Hiroaki Shimizu,Atsuo Kikuchi,Natsuko Arai-Ichinoi,Shigeo Kure,Teiji Tominaga +9 more
TL;DR: Moyamoya disease-deficient mice generated by deleting exon 32 of RNF213 by the Cre-lox system did not spontaneously develop MMD, indicating that a functional loss of R NF213 did not sufficiently induce MMD.
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Pressure-dependent effect of shock waves on rat brain: induction of neuronal apoptosis mediated by a caspase-dependent pathway.
Kaoruko Kato,Miki Fujimura,Atsuhiro Nakagawa,Atsushi Saito,Tomohiro Ohki,Kazuyoshi Takayama,Teiji Tominaga +6 more
TL;DR: High-overpressure shock wave exposure results in brain injury, including neuronal apoptosis mediated by a caspase-dependent pathway, which is the first report in which the pressure-dependent effect of shock wave on the histological characteristics of brain tissue is demonstrated.