M
Miki Fujimura
Researcher at Tohoku University
Publications - 338
Citations - 13058
Miki Fujimura is an academic researcher from Tohoku University. The author has contributed to research in topics: Moyamoya disease & Cerebral blood flow. The author has an hindex of 50, co-authored 289 publications receiving 11341 citations. Previous affiliations of Miki Fujimura include Stanford University.
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Uneven cerebral hemodynamic change as a cause of neurological deterioration in the acute stage after direct revascularization for moyamoya disease: cerebral hyperperfusion and remote ischemia caused by the 'watershed shift'.
Xian kun Tu,Miki Fujimura,Sherif Rashad,Shunji Mugikura,Hiroyuki Sakata,Kuniyasu Niizuma,Teiji Tominaga +6 more
TL;DR: CHP and cerebral infarction may occur simultaneously not only due to blood pressure lowering against CHP, but also to the ‘watershed shift’ phenomenon, which needs to be elucidated in future studies.
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Clinical implications of intraoperative infrared brain surface monitoring during superficial temporal artery-middle cerebral artery anastomosis in patients with moyamoya disease.
TL;DR: Although the present method does not directly monitor surface CBF, temperature rise around the anastomosis site during surgery might be an indicator of postoperative hyperperfusion.
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PTEN gene mutation and high MIB-1 labeling index may contribute to dissemination in patients with glioblastoma.
Hideaki Kato,Miki Fujimura,Toshihiro Kumabe,Chikashi Ishioka,Ryunosuke Kanamaru,Takashi Yoshimoto +5 more
TL;DR: The results indicate that the evaluation of PTEN mutation and MIB-1 LI are useful to predict dissemination and prognosis of glioblastomas.
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Overexpression of Copper-Zinc Superoxide Dismutase Attenuates Acute Activation of Activator Protein-1 After Transient Focal Cerebral Ischemia in Mice
TL;DR: SOD1 overexpression prevents early activation of AP-1 after transient FCI in mice, which may block the expression of downstream target genes that are injurious, thereby reducing the infarction volume after transient AFC in mice.
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Symptomatic hyperperfusion after superficial temporal artery–middle cerebral artery anastomosis in a child with moyamoya disease
TL;DR: It is demonstrated, for the first time, that delayed focal neurological deficit after STA–MCA anastomosis can be caused by focal hyperperfusion in childhood moyamoya disease.