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Mohammad Javad Alemzadeh Ansari

Researcher at University of Tehran

Publications -  13
Citations -  176

Mohammad Javad Alemzadeh Ansari is an academic researcher from University of Tehran. The author has contributed to research in topics: Neuromorphic engineering & Peritoneal dialysis. The author has an hindex of 7, co-authored 13 publications receiving 141 citations. Previous affiliations of Mohammad Javad Alemzadeh Ansari include Tehran University of Medical Sciences.

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An Ultra Low-Power Memristive Neuromorphic Circuit for Internet of Things Smart Sensors

TL;DR: The results of the study reveal that the designed neuromorphic circuits, along with the proposed A/D and D/A converters, provide an average power saving of over the ASIC implementation in a 90-nm CMOS technology.
Journal Article

Survival at 1, 3, and 5 years in diabetic and nondiabetic patients on hemodialysis.

TL;DR: The survival of diabetic patients undergoing hemodialysis was much worse than survival of nondiabetic patients, and prevention of diabetic nephropathy should be more emphasized and other renal replacement therapies such as kidney transplantation must be considered as soon as possible.
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PHAX: Physical Characteristics Aware Ex-Situ Training Framework for Inverter-Based Memristive Neuromorphic Circuits

TL;DR: The results of applying the PHAX approach on the kernels of some applications such as image recognition, image processing, and financial analysis reveal that the designed neuromorphic circuits provide an average power saving over the ASIC implementation in a 90-nm CMOS technology.
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Association between preprocedural hemoglobin level and 1-year outcome of elective percutaneous coronary intervention.

TL;DR: The impact of anemia on the 12-month MACE was more prominent in patients with Hb below 10 mg/dl, and in patientsWith severe anemia listed for PCI, this level of Hb should be considered as a precaution.
Journal Article

ADAMTS-13 deficiency following Hemiscorpius lepturus scorpion sting

TL;DR: The causes of HUS were evaluated and it was found that the levels of C3, C4, CH50 and H factors were normal, but the activity of Von Willebrand factor cleaving protease was decreased (less than 5% of the normal activity).