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Natalia V. Gulyaeva
Researcher at Russian Academy of Sciences
Publications - 267
Citations - 2897
Natalia V. Gulyaeva is an academic researcher from Russian Academy of Sciences. The author has contributed to research in topics: Hippocampus & Hippocampal formation. The author has an hindex of 24, co-authored 241 publications receiving 2334 citations. Previous affiliations of Natalia V. Gulyaeva include Russian Academy & Moscow State University.
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An early decrease in cell proliferation after pentylenetetrazole-induced seizures.
TL;DR: Different temporal profiles of neuronal damage and proliferation changes suggest that neurodegeneration is unlikely to be a global proliferation-regulating factor, and may indicate the existence of global mechanisms affecting cellular proliferation in adult brain during seizures.
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Synaptic vesicle acidification and exocytosis studied with acridine orange fluorescence in rat brain synaptosomes.
TL;DR: AO fluorescence is suitable for monitoring SV acidification within synaptosomes, and may be used to derive an independent estimate of the relative size of the immediately releasable SV pool, which is consistent with depolarization-evoked release of the acidic contents of an exocytosis-competent pool of synaptic vesicles.
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Footshock stress alters early postnatal development of electrophysiological responses and caspase-3 activity in rat hippocampus.
TL;DR: It is suggested that footshock activates the development of hippocampal circuitry during early phases, this phenomenon mediating enhanced responsiveness as a result of an increased production of synaptic connections and related decrease in neuronal loss.
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Glucocorticoid-mediated mechanisms of hippocampal damage: Contribution of subgranular neurogenesis.
TL;DR: Understanding the details of GC‐mediated mechanisms involved in the alterations in AHN could enable the identification of molecular targets for ameliorating pathology‐induced imbalance in the HPA axis/AHN mutual regulation to conquer cognitive and psychiatric disturbances.
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Pentylenetetrazol kindling in rats: Is neurodegeneration associated with manifestations of convulsive activity?
TL;DR: Rats “ tolerant” to developing convulsions show signs of oxidative stress and neurodegenerative changes in the hippocampus, which suggests that oxidative neuron damage leading to neurodegenersation in the pentylenetetrazol kindling model is not directly associated with convulsive activity.