O
Ofer Lider
Researcher at Weizmann Institute of Science
Publications - 106
Citations - 10064
Ofer Lider is an academic researcher from Weizmann Institute of Science. The author has contributed to research in topics: T cell & Extracellular matrix. The author has an hindex of 46, co-authored 106 publications receiving 9803 citations.
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Journal ArticleDOI
Dependence of human stem cell engraftment and repopulation of NOD/SCID mice on CXCR4
Amnon Peled,Isabelle Petit,Orit Kollet,Michal Magid,Tanya Ponomaryov,Tamara Byk,Arnon Nagler,Herzl Ben-Hur,Ariel Many,Leonard D. Shultz,Ofer Lider,Ronen Alon,Dov Zipori,Tsvee Lapidot +13 more
TL;DR: Up-regulation of CXCR4 expression may be useful for improving engraftment of repopulating stem cells in clinical transplantation.
Journal ArticleDOI
The chemokine SDF-1 activates the integrins LFA-1, VLA-4, and VLA-5 on immature human CD34(+) cells: role in transendothelial/stromal migration and engraftment of NOD/SCID mice.
Amnon Peled,Orit Kollet,Tanya Ponomaryov,Isabelle Petit,Suzanna Franitza,Valentin Grabovsky,Michal Magid Slav,Arnon Nagler,Ofer Lider,Ronen Alon,Dov Zipori,Tsvee Lapidot +11 more
TL;DR: It is demonstrated that repopulating human stem cells functionally express LFA-1, VLA-4, andVLA-5, and this study implies a novel approach to further advance clinical transplantation.
Journal ArticleDOI
Suppressor T cells generated by oral tolerization to myelin basic protein suppress both in vitro and in vivo immune responses by the release of transforming growth factor beta after antigen-specific triggering.
TL;DR: It is found that modulator cells from animals orally tolerized to MBP produce a suppressor factor upon stimulation with MBP in vitro that is specifically inhibited by anti-transforming growth factor beta (TGF-beta) neutralizing antibodies.
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Anti-idiotypic network induced by T cell vaccination against experimental autoimmune encephalomyelitis
TL;DR: It is suggested that T cell vaccination induces resistance to autoimmune disease by activating an antiidiotypic network.
Journal ArticleDOI
Association between common Toll-like receptor 4 mutations and severe respiratory syncytial virus disease.
Guy Tal,Avigdor Mandelberg,Ilan Dalal,Ilan Dalal,Karine Cesar,Eli Somekh,Asher Tal,Anat Oron,Svetlana Itskovich,Ami Ballin,Sion Houri,Avraham Beigelman,Ofer Lider,Gideon Rechavi,Ninette Amariglio +14 more
TL;DR: These findings suggest that TLR4 mutations, but not the CD14/-159 polymorphism, are associated with an increased risk of severe RSV bronchiolitis in previously healthy infants.