Øivind Skare

TL;DR: The data suggest that the blood–brain barrier (BBB) is more complex than anticipated, and under certain conditions, the astrocyte covering of brain microvessels is rate limiting to water movement.

TL;DR: Evidence is provided that brain swelling triggers Ca2+ signaling in astrocytes and that deletion of the Aqp4 gene markedly interferes with these events, suggesting that AQP4 not only serves as an influx route for water but also is critical for initiating downstream signaling events that may affect and potentially exacerbate the pathological outcome in clinical conditions associated with brain edema.

TL;DR: An object model for fluvial reservoirs that has been developed from 1985 to present is described in this article using a formal mathematical object model (marked point process) describing the distributions of four facies: channel, crevasse, barrier and background.

TL;DR: It is demonstrated by quantitative immunogold cytochemistry that the ischemic striatum and neocortex show distinct patterns of AQP4 expression in the reperfusion phase after 90 min of middle cerebral artery occlusion, indicating that the size of the AQP 4 pool that controls the exchange of fluid between brain and blood during edema formation and dissolution is subject to large and region-specific changes in the reconstructive phase.

TL;DR: Evaluating the long term effects of the Norwegian School Fruit programme, provided at no-cost to the pupils, three years after it was provided for free shows long-term effects of a free school fruit programme.