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Omar A. Gharbawie

Researcher at Vanderbilt University

Publications -  37
Citations -  1656

Omar A. Gharbawie is an academic researcher from Vanderbilt University. The author has contributed to research in topics: Motor cortex & Posterior parietal cortex. The author has an hindex of 23, co-authored 35 publications receiving 1501 citations. Previous affiliations of Omar A. Gharbawie include University of Lethbridge & University of Pittsburgh.

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Cortical and subcortical plasticity in the brains of humans, primates, and rats after damage to sensory afferents in the dorsal columns of the spinal cord

TL;DR: The anatomical, physiological and behavioral changes that take place in response to injury-induced plasticity after damage to the dorsal column pathway in rats and monkeys are discussed and functional collateral sprouting has been promoted by the post-lesion digestion of the perineuronal net in the cuneate nucleus.
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Thalamocortical Connections of Functional Zones in Posterior Parietal Cortex and Frontal Cortex Motor Regions in New World Monkeys

TL;DR: In owl and squirrel monkeys, long-duration electrical stimulation distinguished several functional zones within the PPC and motor/premotor cortex (M1/PM).
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Multiple parietal-frontal pathways mediate grasping in macaque monkeys

TL;DR: The three grasp zones may be nodes of parallel parietal–frontal pathways that coordinate their respective functions into an accurate grasp, and differential points of origin and termination of each pathway suggest varying functional specializations.
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The exploratory behavior of rats in an open environment optimizes security.

TL;DR: The pattern of behavior, in which the rats failed to explore from a secure starting position and were increasingly likely to run away as security decreased, suggests that a primary function of locomotor behavior in a novel environment is to optimize security.
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Evidence for bilateral control of skilled movements: ipsilateral skilled forelimb reaching deficits and functional recovery in rats follow motor cortex and lateral frontal cortex lesions.

TL;DR: The finding of an enduring ipsilateral impairment in skilled movement is consistent with a large but more anecdotal literature in rats, nonhuman primates and humans, and suggests that plastic changes in the intact hemisphere are related to that hemisphere's contribution to skilled movement.