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Oscar Sans Capdevila

Researcher at Hospital Sant Joan de Déu Barcelona

Publications -  46
Citations -  3731

Oscar Sans Capdevila is an academic researcher from Hospital Sant Joan de Déu Barcelona. The author has contributed to research in topics: Obstructive sleep apnea & Polysomnography. The author has an hindex of 33, co-authored 45 publications receiving 3410 citations. Previous affiliations of Oscar Sans Capdevila include University of Chicago & University of Louisville.

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Metabolic alterations and systemic inflammation in obstructive sleep apnea among nonobese and obese prepubertal children.

TL;DR: The significant improvements in lipid profiles, C-reactive protein, and apolipoprotein B after adenotonsillectomy in the two groups suggest a pathogenic role for OSA in lipid homeostasis and systemic inflammation independent of the degree of adiposity.
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Pediatric Obstructive Sleep Apnea: Complications, Management, and Long-term Outcomes

TL;DR: The clinical prototypic pediatric patient of the early 1990s has been insidiously replaced by a different phenotypic presentation that strikingly resembles that of adults afflicted by the disease, and the terms type I and type II pediatric OSA have been proposed.
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C-reactive Protein, Obstructive Sleep Apnea, and Cognitive Dysfunction in School-aged Children

TL;DR: High-sensitivity C-reactive protein (hsCRP) levels are higher in children with OSA, and particularly in those who develop neurocognitive deficits, suggesting that the magnitude of the inflammatory responses elicited by OSA is a major determinant of increased risk for neuroc cognitive dysfunction.
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Obstructive sleep apnea in children: relative contributions of body mass index and adenotonsillar hypertrophy.

TL;DR: In this article, the authors found that the magnitude of adenotonsillar hypertrophy required for any given magnitude of OAHI is more likely to be smaller in obese children compared to nonobese children.
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Obstructive Sleep Apnea and Endothelial Function in School-Aged Nonobese Children Effect of Adenotonsillectomy

TL;DR: Postocclusive hyperemia is consistently blunted in children with OSA, and such altered endothelial function is reversible 4 to 6 months after treatment, particularly if a family history of cardiovascular disease is not present.