P
P. Fernlund
Researcher at Lund University
Publications - 5
Citations - 292
P. Fernlund is an academic researcher from Lund University. The author has contributed to research in topics: Diabetes mellitus & Antibody. The author has an hindex of 5, co-authored 5 publications receiving 291 citations.
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Journal ArticleDOI
β-Cell Function in Relation to Islet Cell Antibodies During the First 3 Yr After Clinical Diagnosis of Diabetes in Type II Diabetic Patients
TL;DR: In patients considered type II diabetic with ICA, β-cell function progressively decreased after diagnosis, and after 3 yr was similar to type I diabetic patients, whereas β- cell function intype II diabetic patients without ICA was unchanged.
Journal ArticleDOI
Glutamate decarboxylase antibody levels predict rate of β-cell decline in adult-onset diabetes
Anders Gottsäter,Mona Landin-Olsson,Åke Lernmark,Åke Lernmark,P. Fernlund,G. Sundkvist,W. A. Hagopian +6 more
TL;DR: Not only did G AD65Ab presence predict future insulin dependence, but higher GAD65Ab levels may mark more rapid decline in beta-cell function in apparent non-insulin-dependent diabetes.
Journal ArticleDOI
Autonomic neuropathy in Type 2 diabetic patients is associated with hyperinsulinaemia and hypertriglyceridaemia
TL;DR: Aims to clarify whether parasympathetic neuropathy in Type 2 diabetic patients is associated with features of the insulin resistance syndrome.
Journal ArticleDOI
Islet cell antibodies are associated with β-cell failure also in obese adult onset diabetic patients
TL;DR: ICA detected at diagnosis of diabetes in both obese and nonobese adult patients indicate β-cell dysfunction, high HbA1c levels, and progression to insulin dependency.
Journal ArticleDOI
Pancreatic beta-cell function evaluated by intravenous glucose and glucagon stimulation. A comparison between insulin and C-peptide to measure insulin secretion
TL;DR: C-peptide measurements in healthy subjects are more reproducible than insulin measurements in determination of beta-cell function, showing no significant differences in median within-subject variation between the initial insulin and C- peptide responses to glucose.