P
P. Landry
Researcher at McGill University
Publications - 7
Citations - 607
P. Landry is an academic researcher from McGill University. The author has contributed to research in topics: Somatosensory system & Receptive field. The author has an hindex of 7, co-authored 7 publications receiving 601 citations. Previous affiliations of P. Landry include University of Tennessee.
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Journal ArticleDOI
Functional role of GABA in cat primary somatosensory cortex: shaping receptive fields of cortical neurons.
TL;DR: A differential effect of BMI is suggested, suggesting that GABA controls receptive-field size in rapidly adapting regions, and indicates that neurons in rapid adapting regions differ pharmacologically from those in other submodality regions.
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Morphological and electrophysiological characteristics of pyramidal tract neurons in the rat.
TL;DR: Responses evoked in neurons of rat sensorimotor cortex upon stimulation of the pyramidal tract and ipsilateral cerebral peduncle were analysed using intracellular recording and no cells comparable to the fast conducting PT cells observed in other species were found.
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Bicuculline-induced alterations of response properties in functionally identified ventroposterior thalamic neurones.
TL;DR: Modality and submodality properties of all cells tested apparently remained unchanged qualitatively, despite all pharmacological interventions, and BMI changed the temporal properties of the responses of both thalamocortical relay neurones and of presumed interneurones.
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Somatosensory cortical neurons with an identifiable electrophysiological signature.
TL;DR: In both cats and rats, neurons with a distinctively narrow action potential were recognized as a small subset of all neurons isolated in the somatosensory cortex, suggesting that there may be more than one class of cortical neurons with thin spikes.
Journal ArticleDOI
Identification of two populations of corticothalamic neurons in cat primary somatosensory cortex
P. Landry,Robert W. Dykes +1 more
TL;DR: It is suggested that only type 2 CT cells can modify the activity of thalamic neurons through a corticothalamic feedback loop.