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P. N. Samuelson

Researcher at University of Alabama at Birmingham

Publications -  28
Citations -  1363

P. N. Samuelson is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Vascular resistance & Hemodynamics. The author has an hindex of 15, co-authored 28 publications receiving 1341 citations. Previous affiliations of P. N. Samuelson include University of Alabama.

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Neuronal and adrenomedullary catecholamine release in response to cardiopulmonary bypass in man.

TL;DR: A significant increase in catecholamines could jeopardize myocardial protective measures during CPB, and the predominant humoral response to CPB appears to be adrenomedullary release of E.
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Hemodynamic Responses to Anesthetic Induction with Midazolam or Diazepam in Patients with Ischemic Heart Disease

TL;DR: Although differing somewhat, midazolam, like diazepam, provided rapid, hemodynamically stable induction of anesthesia in patients with ischemic heart disease, and it was found that patients in both groups responded to endotracheal intubation with transient increases in blood pressure, heart rate, and systemic vascular resistance, but the hemodynamic values spontaneously returned toward control levels within 2 to 5 minutes.
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Regional blood flow during cross-clamping of the thoracic aorta and infusion of sodium nitroprusside.

TL;DR: The data show that SNP infusion during thoracic aortic cross-clamping improves systemic and regional circulation above the occlusion but decreases MAPb and therefore blood flow below the Occlusion, which suggests that blood flow through organs and tissues below theOcclusion is pressure dependent.
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Relationship of whole body oxygen consumption to perfusion flow rate during hypothermic cardiopulmonary bypass.

TL;DR: During cardiopulmonary bypass cerebral blood flow, autoregulation seems present at 20 degrees C, and the data set indicates that flows of about 1.2 may be adequate despite limited reserves.
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Cardiovascular effects of esmolol in anesthetized humans.

TL;DR: It is shown that esmolol can be used safely during anesthesia in patients with normal ventricular function to attenuate cardiac response to sympathetic stimulation and antagonizes the β-adrenergic effects of norepinephrine.