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Pamela J. VandeVord
Researcher at Virginia Tech
Publications - 105
Citations - 3770
Pamela J. VandeVord is an academic researcher from Virginia Tech. The author has contributed to research in topics: Traumatic brain injury & Poison control. The author has an hindex of 32, co-authored 93 publications receiving 3417 citations. Previous affiliations of Pamela J. VandeVord include United States Department of Veterans Affairs & Wake Forest University.
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Journal ArticleDOI
Evaluation of the biocompatibility of a chitosan scaffold in mice.
Pamela J. VandeVord,Howard W. T. Matthew,Stephen P. DeSilva,Lois Mayton,Bin Wu,Paul H. Wooley +5 more
TL;DR: It is suggested that chitosan may be suitable for the development of implantable materials after a preliminary study showed a high degree of biocompatibility in this animal model.
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Improved tissue-engineered bone regeneration by endothelial cell mediated vascularization.
TL;DR: The results indicated that the integration of complex cell populations with composite scaffold materials provided an effective technique to improve osteogenesis in engineered bone grafts, suggesting that hybrid grafts have great potential for clinical use to treat large bone defects.
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A modular approach for the generation, storage, mixing, and detection of droplet libraries for high throughput screening
TL;DR: Due to its flexibility, the widespread availability of components, and some favorable material properties compared to PDMS, this approach can be a useful part of a researcher's toolkit for prototyping droplet-based assays.
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Blast induces oxidative stress, inflammation, neuronal loss and subsequent short-term memory impairment in rats.
Hyung Joon Cho,Venkata Siva Sai Sujith Sajja,Pamela J. VandeVord,Pamela J. VandeVord,Yong Woo Lee +4 more
TL;DR: Pro-oxidative and pro-inflammatory environments in the brain could play a potential role in BOP-induced neuronal loss and behavioral deficits and may provide a foundation for defining a molecular and cellular basis of the pathophysiology of blast-induced neurotrauma (BINT).
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Intracranial pressure increases during exposure to a shock wave
TL;DR: It is demonstrated that proper sealing techniques lead to a significant increase in ICP values, compared to the outside overpressure generated by the blast, which suggests that a global flexure of the skull by the transient shockwave is an important mechanism of pressure transmission inside the brain.