P
Paul I. Korner
Researcher at Alfred Hospital
Publications - 122
Citations - 5482
Paul I. Korner is an academic researcher from Alfred Hospital. The author has contributed to research in topics: Blood pressure & Baroreceptor. The author has an hindex of 38, co-authored 122 publications receiving 5419 citations. Previous affiliations of Paul I. Korner include Baker IDI Heart and Diabetes Institute & Royal Prince Alfred Hospital.
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Journal ArticleDOI
Norepinephrine spillover to plasma in patients with congestive heart failure: evidence of increased overall and cardiorenal sympathetic nervous activity.
TL;DR: There is marked regional variation, inapparent from measurements of plasma norepinephrine concentration, in sympathetic nerve activity in patients with congestive heart failure.
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Determination of norepinephrine apparent release rate and clearance in humans.
Murray D. Esler,G. Jackman,Alex Bobik,Dianne Kelleher,Garry L. Jennings,P. Leonard,H. Skews,Paul I. Korner +7 more
TL;DR: The findings suggest that methods which measure the dynamic processes of norepinephrine release and removal quantify sympathetic nervous activity better than steady state plasma norpinephrine measurements alone.
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‘Steady-state’ properties of the baroreceptor-heart rate reflex in essential hypertension in man
TL;DR: Rises and falls in mean arterial and pulse pressures from the resting value were evoked by intravenous injections of phenylephrine and glyceryl trinitrate, and were related to the reflexly evoked changes in heart period (HP; pulse interval).
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Reflex and mechanical circulatory effects of graded Valsalva maneuvers in normal man
TL;DR: The afferent input profile is probably complex, and the role of the different receptor groups may vary at the different levels of EP.
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The renal sympathetic baroreflex in the rabbit. Arterial and cardiac baroreceptor influences, resetting, and effect of anesthesia.
TL;DR: During resettingassociated with nitroprusside-induced falls in resting blood pressure, high-gain reflex adjustments in renal sympathetic nerve activity to moderate changes in mean arterial pressure were preserved, but during resetting associated with phenylephrine-induced rises in resting mean mean arterials pressure, the resting renal sympathetic nerves activity lay on the lower curve plateau, resulting in reduction in the apparent gain of the reflex.