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Paulami Naik

Researcher at Tufts Center for the Study of Drug Development

Publications -  8
Citations -  881

Paulami Naik is an academic researcher from Tufts Center for the Study of Drug Development. The author has contributed to research in topics: Medicine & Pharmacy. The author has an hindex of 5, co-authored 5 publications receiving 179 citations. Previous affiliations of Paulami Naik include Massachusetts Institute of Technology & Claremont Colleges.

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Journal ArticleDOI

Estimating excess mortality due to the COVID-19 pandemic: a systematic analysis of COVID-19-related mortality, 2020–21

Haidong Wang, +95 more
- 01 Mar 2022 - 
TL;DR: It is estimated that 18·2 million people died worldwide because of the COVID-19 pandemic (as measured by excess mortality) over that period, and the number of excess deaths was largest in the regions of south Asia, north Africa and the Middle East, and eastern Europe.
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Early (M170) activation of face-specific cortex by face-like objects

TL;DR: This article found that objects incidentally perceived as faces evoked an early activation in the ventral fusiform cortex, at a time and location similar to that evoked by faces, whereas common objects did not evoke such activation.
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Pandemic preparedness and COVID-19: an exploratory analysis of infection and fatality rates, and contextual factors associated with preparedness in 177 countries, from Jan 1, 2020, to Sept 30, 2021

TL;DR: High levels of government and interpersonal trust, as well as less government corruption, were also associated with higher COVID-19 vaccine coverage among middle-income and high-income countries where vaccine availability was more widespread, and lower corruption was associated with greater reductions in mobility.
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Indecision in Neural Decision Making Models

TL;DR: The dynamics of a model of two interacting neural populations with mutual time–delayed inhibition is examined, finding that when the input to each population is sufficiently high, there is bistability and the dynamics is determined by the relationship of the initial function to the separatrix.
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Is migraine a lateralization defect

TL;DR: The data suggest that migraine's circadian component and its association with PFO may be linked to a lateralization defect during embryogenesis, which could be a result from abnormal serotonin regulation.