Peter Biberfeld

TL;DR: A novel human B-lymphotropic virus (HBLV) was isolated from the peripheral blood leukocytes of six individuals and selectively infected freshly isolated human B cells and converted them into large, refractile mono- or binucleated cells with nuclear and cytoplasmic inclusion bodies.

TL;DR: It is shown that the AIDS-KS cells express messenger RNA for a complex mixture of cytokines that correlate with several of the biological activities of these cells, which seem to account for a large proportion of the activities.

TL;DR: It is demonstrated that KSHV-FLIP protects cells from Fas-mediated apoptosis by inhibiting caspase activation and permits clonal growth in the presence of death stimuli in vitro, and it is shown that it can act as a tumor progression factor by promoting tumor establishment and growth in vivo.

TL;DR: The capacity of MO and even of Mϕ to differentiate into DC was conserved for at least this period and the increased capacity to stimulate the mixed leukocyte reaction correlated with the relative number of CD1a− cells under each condition tested, a confirmation that these cells functionally qualify as DC.

TL;DR: It is suggested that the presence of novel, herpes virus DNA sequences in more than 90% of AIDS Kaposi lesions (AKS) would strongly suggest a specific, aetiopathological involvement of this putative new herpes virus in the pathogenesis of Kaposi's sarcoma, rather than a contamination of yet another opportunistic virus in immunosuppressed AIDS patients.