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Peter C. Konturek

Researcher at University of Jena

Publications -  180
Citations -  5983

Peter C. Konturek is an academic researcher from University of Jena. The author has contributed to research in topics: Helicobacter pylori & Gastric mucosa. The author has an hindex of 42, co-authored 175 publications receiving 5374 citations. Previous affiliations of Peter C. Konturek include University of Erlangen-Nuremberg.

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Continuous Positive Airway Pressure Treatment Rapidly Improves Insulin Sensitivity in Patients with Obstructive Sleep Apnea Syndrome

TL;DR: The improved insulin sensitivity after 2 nights of treatment may reflect a decreasing sympathetic activity, indicating that sleep apnea is an independent risk factor for increased insulin resistance.
Journal Article

Stress and the gut: pathophysiology, clinical consequences, diagnostic approach and treatment options

TL;DR: In this paper, the authors defined stress as an acute threat to homeostasis, and showed both short and long-term effects on the functions of the gastrointestinal tract, which can lead to the development of a broad array of gastrointestinal disorders including inflammatory bowel disease (IBD), irritable bowel syndrome (IBS), and other functional gastrointestinal diseases, food antigen-related adverse responses, peptic ulcer and gastroesophageal reflux disease (GERD).
Journal Article

Gut clock: implication of circadian rhythms in the gastrointestinal tract

TL;DR: Identification of the molecular basis and role of melatonin in the regulation of circadian rhythm allows researchers and clinicians to approach gastrointestinal diseases from a chronobiological perspective.
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The Effect of Continuous Positive Airway Pressure Treatment on Insulin Sensitivity in Patients with Obstructive Sleep Apnoea Syndrome and Type 2 Diabetes

TL;DR: The results indicate that OSA itself is an independent risk factor for insulin resistance, and this effect may be explained by the elevated sympathetic activity in OSA.
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Antrum- and corpus mucosa-infiltrating CD4(+) lymphocytes in Helicobacter pylori gastritis display a Th1 phenotype.

TL;DR: CD4+ cells infiltrating antrum and corpus stomach mucosa during H. pylori infection show a Th1 phenotype, and this polarized Th1-type response may contribute to the inability of the immune system to eradicate the bacterium.