P
Peter Feindt
Researcher at University of Düsseldorf
Publications - 120
Citations - 2171
Peter Feindt is an academic researcher from University of Düsseldorf. The author has contributed to research in topics: Extracorporeal circulation & Aortic valve replacement. The author has an hindex of 27, co-authored 117 publications receiving 2061 citations.
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Increased preoperative C-reactive protein (CRP)-values without signs of an infection and complicated course after cardiopulmonary bypass (CPB)-operations
TL;DR: Data show that patients without apparent infection or inflammation, who had elevated CRP-values preoperatively, face an increased risk of septic complications after extracorporeal circulation.
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Pulsatile extracorporeal circulation during on-pump cardiac surgery enhances aortic wall shear stress.
Alexander Assmann,Ali Cemal Benim,Fethi Gül,Philipp Lux,Payam Akhyari,Udo Boeken,Franz Joos,Peter Feindt,Artur Lichtenberg +8 more
TL;DR: Evaluating the aortic blood flow and wall shear stress profiles under the influence of antegrade or retrograde perfusion with pulsatile versus non-pulsatile extracorporeal circulation finds that a pulsatile perfusion extensively enhanced the occurrence of turbulences, maximum blood flow speed and maximum wallShear stress.
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Intraaortic balloon pumping in patients with right ventricular insufficiency after cardiac surgery: parameters to predict failure of IABP Support.
TL;DR: In patients with IABP support for postcardiotomy cardiogenic shock, elevated serum lactate, elevated base deficit, hypotension, oliguria and large vasopressor doses are all predictors of mortality.
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Heart-type fatty acid binding protein (hFABP) in the diagnosis of myocardial damage in coronary artery bypass grafting.
TL;DR: In this article, the authors used heart-type fatty acid binding protein (hFABP) as a rapid marker of perioperative myocardial damage and peaks earlier than CKMB or TnI.
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Markers of Activated Hemostasis and Fibrinolysis in Patients with Pulmonary Malignancies: Comparison of Plasma Levels in Central Venous and Pulmonary Venous Blood
TL;DR: It is indicated that malignant lung tumors directly contribute to the activation of hemostasis and fibrinolysis in these clinical settings.