P
Piotr Bregestovski
Researcher at French Institute of Health and Medical Research
Publications - 52
Citations - 7030
Piotr Bregestovski is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Glycine receptor & NMDA receptor. The author has an hindex of 32, co-authored 50 publications receiving 6818 citations. Previous affiliations of Piotr Bregestovski include University College London & Pasteur Institute.
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Calcium and endocannabinoids in the modulation of inhibitory synaptic transmission
TL;DR: Both postsynaptic and presynaptic mechanisms mediating changes in synaptic strength will be examined, and particular attention will be devoted to phenomena of retrograde signaling and the recently discovered role, played by the endocannabinoid system in retrograde synaptic modulation.
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Isolation and characterization of an alpha 2-type zebrafish glycine receptor subunit.
TL;DR: Analysis of messenger RNA revealed that alphaZ2 is specifically expressed in adult brain tissue and present in both adult and embryonic zebrafish, contributing to the characterization of the diversity of glycine receptor isoforms in vertebrates.
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Reactive Oxygen Species Initiate a Metabolic Collapse in Hippocampal Slices: Potential Trigger of Cortical Spreading Depression
Anton Malkov,Anton Ivanov,Irina Popova,Marat Mukhtarov,Olena Gubkina,Tatsiana Waseem,Piotr Bregestovski,Yuri Zilberter +7 more
TL;DR: It is found that Tempol strongly reduced occurrence of CSD in vivo, suggesting that ROS accumulation may be a key mechanism ofCSD initiation.
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Regulation of potassium conductance in the cellular membrane at early embryogenesis.
TL;DR: It is proposed that oscillations of the resting potential during the cell-cleavage cycle arise due to modulation of SA channel sensitivity to stretch through cAMP-dependent phosphorylation.
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Inhibition of spontaneous network activity in neonatal hippocampal slices by energy substrates is not correlated with intracellular acidification.
TL;DR: In this paper, the authors investigated the effect of complementary energy substrates (CES) on the neuronal properties governing excitability in neonatal brain slices, and found that the effect is not caused by a subtle intracellular acidification induced by these compounds, but by modulation of neuronal energy homeostasis.