P
Priyanka Trivedi
Researcher at University of Oulu
Publications - 44
Citations - 1379
Priyanka Trivedi is an academic researcher from University of Oulu. The author has contributed to research in topics: Oxidative stress & DNA damage. The author has an hindex of 20, co-authored 41 publications receiving 1055 citations. Previous affiliations of Priyanka Trivedi include Brigham and Women's Hospital & Saurashtra University.
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Oxidative stress in ulcerative colitis: an old concept but a new concern.
TL;DR: The role of oxidative stress in the pathogenesis of ulcerative colitis and the associated local as well as global DNA damage, which may lead to carcinogenesis is portrayed.
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Cardioprotective effects of hesperetin against doxorubicin-induced oxidative stress and DNA damage in rat.
TL;DR: Hsperetin ameliorated doxorubicin-induced cardiotoxicity by reducing oxidative stress, abnormal cellular morphology and DNA damage in rat, and nuclear factor-kappa B, p38, and caspase-3 play a role in the he Speretin-mediated protection against dox orubicIn-inducedCardiotoxicity.
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Melatonin modulated autophagy and Nrf2 signaling pathways in mice with colitis-associated colon carcinogenesis.
TL;DR: MEL intervention reduced autophagy by ameliorating inflammation and oxidative stress in the colon of mice with CACC, and attenuates the progression of CACC in mice by modulating autophileagy and Nrf2 signaling pathways.
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Developmental and Environmental Regulation of Cuticular Wax Biosynthesis in Fleshy Fruits
Priyanka Trivedi,Nga Nguyen,Anne Linn Hykkerud,Hely Häggman,Inger Martinussen,Laura Jaakola,Katja Karppinen +6 more
TL;DR: This review focuses on the current knowledge of the cuticular wax biosynthesis during fleshy fruits development, and on the effect of environmental factors in regulation of the biosynthesis.
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Investigation on sodium valproate induced germ cell damage, oxidative stress and genotoxicity in male Swiss mice.
Sabbir Khan,Tauseef Ahmad,Chintan Vishnubhai Parekh,Priyanka Trivedi,Sapana Kushwaha,Gopabandhu Jena +5 more
TL;DR: VPA treatment significantly decreased the sperm count, testes and epididymis weight and significantly increased the sperm head abnormality, sperm DNA damage, oxidative stress and 8-oxo-dG expression in the testes of mice.