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Qinghui Mu

Researcher at Virginia–Maryland Regional College of Veterinary Medicine

Publications -  17
Citations -  1566

Qinghui Mu is an academic researcher from Virginia–Maryland Regional College of Veterinary Medicine. The author has contributed to research in topics: Systemic lupus erythematosus & Gut flora. The author has an hindex of 10, co-authored 15 publications receiving 939 citations. Previous affiliations of Qinghui Mu include Virginia Tech & Stanford University.

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Role of Lactobacillus reuteri in human health and diseases

TL;DR: Inflammatory diseases, including those located in the gut as well as in remote tissues, may be ameliorated by increasing the colonization of L. reuteri, which may be an attractive preventive and/or therapeutic avenue against inflammatory diseases.
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Leaky Gut As a Danger Signal for Autoimmune Diseases

TL;DR: It is hypothesized that modulating the gut microbiota can serve as a potential method for regulating intestinal permeability and may help to alter the course of autoimmune diseases in susceptible individuals.
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Control of lupus nephritis by changes of gut microbiota

TL;DR: These beneficial effects were present in female and castrated male mice, but not in intact males, suggesting that the gut microbiota controls lupus nephritis in a sex hormone-dependent manner, and demonstrates essential mechanisms on how changes of the Gut microbiota regulate l upus-associated immune responses in mice.
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Gut Microbiota in Human Systemic Lupus Erythematosus and a Mouse Model of Lupus

TL;DR: Results of the human study suggest that, compared to control subjects without immune-mediated diseases, SLE patients with active lupus disease possessed an altered gut microbiota that differed in several particular bacterial species and was less diverse, with increased representation of Gram-negative bacteria.
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SLE: Another Autoimmune Disorder Influenced by Microbes and Diet?

TL;DR: The available evidence on the contributions of diet and gut microbes to SLE occurrence and pathogenesis is compiled to shed light on how this autoimmune disorder develops, and provide opportunities for improved biomarkers of the disease and the potential to probe new therapies.