Q
Qingwen Xie
Researcher at Wuhan University
Publications - 12
Citations - 178
Qingwen Xie is an academic researcher from Wuhan University. The author has contributed to research in topics: Cardiac fibrosis & Internal medicine. The author has an hindex of 5, co-authored 9 publications receiving 62 citations.
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Journal ArticleDOI
Andrographolide Protects Against Adverse Cardiac Remodeling After Myocardial Infarction through Enhancing Nrf2 Signaling Pathway.
Saiyang Xie,Wei Deng,Jiao-Jiao Chen,Qing-Qing Wu,Hongjian Li,Juan Wang,Li Wei,Chen Liu,Mingxia Duan,Zhulan Cai,Qingwen Xie,Tongtong Hu,Xiao-Feng Zeng,Qi-Zhu Tang +13 more
TL;DR: It is suggested that Andr alleviates adverse cardiac remodeling following myocardial infarction through enhancing Nrf2 signaling pathway.
Journal ArticleDOI
High-mobility group AT-hook 1 promotes cardiac dysfunction in diabetic cardiomyopathy via autophagy inhibition.
Qing-Qing Wu,Chen Liu,Zhulan Cai,Qingwen Xie,Tongtong Hu,Mingxia Duan,Hai-Ming Wu,Yuan Yuan,Qi-Zhu Tang +8 more
TL;DR: The data indicate that HMGA1 aggravates diabetic cardiomyopathy by directly regulating miR-222 promoter activity, which inhibits P27/mTOR-induced autophagy.
Journal ArticleDOI
Indigo fruits ingredient, aucubin protects against LPS-induced cardiac dysfunction in mice.
Ming Xia Duan,Yuan Yuan,Chen Liu,Zhulan Cai,Qingwen Xie,Tongtong Hu,Qi-Zhu Tang,Qing-Qing Wu +7 more
TL;DR: Aucubin exerts a protective effect against lipopolysaccharide-induced cardiac dysfunction by regulating nucleotide-binding oligomerization domain-like receptor family pyrin domain-containing 3 inflammasome and inactivating the NLRP3/ASC/caspase-1 inflam Masome.
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S100A8/A9 in Myocardial Infarction: A Promising Biomarker and Therapeutic Target.
TL;DR: This review focuses on the molecular mechanisms of S100A8/A9 in MI pathogenesis to provide a promising biomarker and therapeutic target for MI.
Journal ArticleDOI
The effect of HMGA1 in LPS-induced Myocardial Inflammation.
Zhulan Cai,Bo Shen,Yuan Yuan,Chen Liu,Qingwen Xie,Tongtong Hu,Qi Yao,Qing-Qing Wu,Qi-Zhu Tang +8 more
TL;DR: It is suggested that overexpression of HMGA1 aggravated cardiomyocytes inflammation and apoptosis by up-regulating COX-2 expression, while silence ofHMGA1 expression attenuated inflammation but aggregated cell apoptosis via down-regulation of STAT3.