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R. Wayne Alexander

Researcher at Emory University

Publications -  103
Citations -  16346

R. Wayne Alexander is an academic researcher from Emory University. The author has contributed to research in topics: Angiotensin II & Vascular smooth muscle. The author has an hindex of 49, co-authored 100 publications receiving 15601 citations. Previous affiliations of R. Wayne Alexander include Merck & Co. & Emory University Hospital.

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Journal ArticleDOI

Markers of Inflammation and Cardiovascular Disease Application to Clinical and Public Health Practice: A Statement for Healthcare Professionals From the Centers for Disease Control and Prevention and the American Heart Association

Thomas A. Pearson, +14 more
- 28 Jan 2003 - 
TL;DR: There has been no consensus from professional societies or governmental agencies as to how these assays of markers of inflammation should be used in clinical practice, and a workshop to address these issues was convened in Atlanta, Ga.
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p38 Mitogen-activated Protein Kinase Is a Critical Component of the Redox-sensitive Signaling Pathways Activated by Angiotensin II ROLE IN VASCULAR SMOOTH MUSCLE CELL HYPERTROPHY

Masuko Ushio-Fukai, +3 more
- 12 Jun 1998 - 
TL;DR: Results provide the first evidence that p38MAPK is a critical component of the oxidant stress (H2O2)-sensitive signaling pathways activated by angiotensin II in vascular smooth muscle cells and indicate that it plays a crucial role in vascular hypertrophy.
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Elevation of C-reactive protein in "active" coronary artery disease.

Bradford C. Berk, +5 more
- 15 Jan 1990 - 
TL;DR: It is suggested that an inflammatory component in "active" angina may contribute to the susceptibility of patients with unstable angina to vasospasm and thrombosis.
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A randomized, double-blinded, placebo-controlled multicenter trial of adenosine as an adjunct to reperfusion in the treatment of acute myocardial infarction (AMISTAD-II)

Allan M. Ross, +4 more
- 07 Jun 2005 - 
TL;DR: Clinical outcomes in patients with STEMI undergoing reperfusion therapy were not significantly improved with adenosine, although infarct size was reduced with the 70-microg/kg/min adenosines infusion, a finding that correlated with fewer adverse clinical events.
Journal ArticleDOI

Reactive Oxygen Species Mediate the Activation of Akt/Protein Kinase B by Angiotensin II in Vascular Smooth Muscle Cells

Masuko Ushio-Fukai, +6 more
- 06 Aug 1999 - 
TL;DR: The studies indicate that Akt/PKB is part of the remarkable spectrum of angiotensin II signaling pathways and provide insight into the highly organized signaling mechanisms coordinated by ROS, which mediate the hypertrophic response to angiotENSin II in VSMCs.