Rachel R. Markwald

TL;DR: Evidence is provided that sleep plays a key role in energy metabolism and increased food intake during insufficient sleep is a physiological adaptation to provide energy needed to sustain additional wakefulness; yet when food is easily accessible, intake surpasses that needed.

TL;DR: It is demonstrated that evening caffeine consumption delays the human circadian melatonin rhythm in vivo and that chronic application of caffeine lengthens the circadian period of molecular oscillations in vitro, primarily with an adenosine receptor/cyclic adenosin monophosphate (AMP)–dependent mechanism.

TL;DR: Findings indicate morning wakefulness and eating during the biological night is a novel mechanism by which short sleep duration contributes to metabolic dysregulation and suggests food intake during the Biological night may contribute to other health problems associated with shortSleep duration.

TL;DR: In this paper, the performance of consumer sleep-tracking devices, alongside actigraphy, was compared with the gold-standard sleep assessment technique, polysomnography (PSG).

TL;DR: The collective findings indicate that mechanical influences contribute largely to the immediate vasodilatation (first cardiac cycle) observed in response to a brief, single contraction, however, it is clear that there are additional mechanisms related to muscle activation that continue to cause and sustain vasodILatation for several more cardiac cycles after contraction.