R
Ralph I. Horwitz
Researcher at Temple University
Publications - 30
Citations - 1070
Ralph I. Horwitz is an academic researcher from Temple University. The author has contributed to research in topics: Evidence-based medicine & MEDLINE. The author has an hindex of 13, co-authored 30 publications receiving 911 citations. Previous affiliations of Ralph I. Horwitz include New York Academy of Medicine & Case Western Reserve University.
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Journal ArticleDOI
Applying results of randomised trials to clinical practice: impact of losses before randomisation.
TL;DR: A survey of 41 clinical trials listed in the 1979 inventory of the National Institute of Health (NINTH) showed that only 10-14% of the patients who were eligible but not entered were similar demographically but differed clinically from those enrolled.
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Major Risk Factors for Intracerebral Hemorrhage in the Young Are Modifiable
Edward Feldmann,Joseph P. Broderick,Walter N. Kernan,Catherine M. Viscoli,Lawrence M. Brass,Thomas G. Brott,Lewis B. Morgenstern,Janet Wilterdink,Ralph I. Horwitz +8 more
TL;DR: Among young men and women, the major risk factors for primary ICH can be modified, suggesting that this type of stroke may be preventable, and findings for caffeine and menopause warrant further study.
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Beyond randomised versus observational studies
John Concato,Ralph I. Horwitz +1 more
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Estrogen therapy and risk of cognitive decline: results from the Women's Estrogen for Stroke Trial (WEST)
Catherine M. Viscoli,Lawrence M. Brass,Walter N. Kernan,Philip M. Sarrel,Samy Suissa,Ralph I. Horwitz +5 more
TL;DR: In this article, the authors conducted a randomized, double-blind trial of estradiol 17β versus placebo for secondary stroke prevention in 664 postmenopausal women with a recent stroke or transient ischemic attack.
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Depressed autonomic nervous system function in African Americans and individuals of lower social class: a potential mechanism of race- and class-related disparities in health outcomes.
TL;DR: Heart rate variability is lower in African Americans and individuals of lower social class, independent of the effects of measured clinical, psychological, or behavioral factors, which suggests that the adverse effects of minority race and lower socialclass on cardiovascular outcomes may be mediated by dysregulation of autonomic function.