R
Ram Singh
Researcher at University of California, Los Angeles
Publications - 145
Citations - 4992
Ram Singh is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Systemic lupus erythematosus & Autoantibody. The author has an hindex of 41, co-authored 133 publications receiving 4573 citations. Previous affiliations of Ram Singh include University of Cincinnati & All India Institute of Medical Sciences.
Papers
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Journal ArticleDOI
A role for sex chromosome complement in the female bias in autoimmune disease.
Deborah L. Smith-Bouvier,Anagha A. Divekar,Manda V. Sasidhar,Sienmi Du,Seema K. Tiwari-Woodruff,Jennifer K. King,Arthur P. Arnold,Ram Singh,Rhonda R. Voskuhl +8 more
TL;DR: This study determined if there was a contribution of sex chromosomes to sex differences in susceptibility to two immunologically distinct disease models, experimental autoimmune encephalomyelitis and pristane-induced lupus.
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TNFα blockade in human diseases: Mechanisms and future directions
Maida Wong,David Ziring,Yael Korin,Sheetal B Desai,Sungjin Kim,Jan Lin,David W. Gjertson,Jonathan Braun,Elaine F. Reed,Ram Singh +9 more
TL;DR: Focusing on the human therapeutic experience, this analytical review will review the biology of mechanisms of action, the limiting factors contributing to disease restriction in therapeutic efficacy, and the mechanism and frequency of treatment-limiting adverse responses of TNFα antagonists.
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TNFα blockade in human diseases : An overview of efficacy and safety
Jan Lin,David Ziring,Sheetal B Desai,Sungjin Kim,Maida Wong,Yael Korin,Jonathan Braun,Elaine F. Reed,David W. Gjertson,Ram Singh +9 more
TL;DR: These two review articles are hoped to stimulate a fresh assessment of the priorities for research and clinical innovation to improve and extend therapeutic use and safety of TNFα antagonism.
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Prevalence of rheumatoid arthritis in the adult Indian population
TL;DR: The prevalence of rheumatoid arthritis in India is quite similar to that reported from the developed countries, and it is higher than those reported from China, Indonesia, Philippines and rural Africa.
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Neonatal peptide exposure can prime T cells and, upon subsequent immunization, induce their immune deviation: implications for antibody vs. T cell-mediated autoimmunity.
TL;DR: Neonatal exposure to antigen is believed to result in T cell clonal inactivation or deletion, but it is reported that, contrary to this notion, neonatal injection of BALB/c mice with a hen egg lysozyme peptide 106- 116 in putative "tolergenic" doses induced a T cell proliferative and an immunoglobulin G (IgG) antibody (Ab) response of both T helper cell 1 and Th2-dependent isotypes.