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Renaud Tamisier

Researcher at University of Grenoble

Publications -  343
Citations -  7770

Renaud Tamisier is an academic researcher from University of Grenoble. The author has contributed to research in topics: Obstructive sleep apnea & Sleep apnea. The author has an hindex of 43, co-authored 296 publications receiving 6051 citations. Previous affiliations of Renaud Tamisier include French Institute of Health and Medical Research & Core Laboratories.

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14 nights of intermittent hypoxia elevate daytime blood pressure and sympathetic activity in healthy humans

TL;DR: These data are the first to show that the arterial pressure rise is sustained throughout the waking hours beyond the acute phase immediately after exposure, and may suggest that sympathoactivation induced by IH likely contributes to blood pressure elevation and may derive from reduced baroreflex inhibition.
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Mechanisms of cardiac dysfunction in obstructive sleep apnea.

TL;DR: The gold-standard treatment for OSA, nasal continuous positive airway pressure (CPAP), might improve cardiac symptoms and hemodynamic parameters in patients with the disease, however, large clinical trials are required to improve the understanding of the cardiac consequences of OSA.
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Comparison of continuous positive airway pressure and valsartan in hypertensive patients with sleep apnea.

TL;DR: In an RCT, although the BP decrease was significant with CPAP treatment, valsartan induced a fourfold higher decrease in mean 24-hour BP than CPAP in untreated hypertensive patients with OSA.
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Intermittent hypoxia and sleep-disordered breathing: current concepts and perspectives

TL;DR: Overall, chronic intermittenthypoxia appears to have specific genomic effects that differ notably from continuous hypoxia, and further research is required to fully elucidate the molecular and cellular mechanisms.
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Hypertension and sleep: overview of a tight relationship.

TL;DR: In the present report, sleep problems/disorders, which impact either the quality or quantity of sleep, are reviewed for their interaction with BP regulation and their potential association with prevalent or incident hypertension.