R
Ricardo Vaz Breda
Researcher at Pontifícia Universidade Católica do Rio Grande do Sul
Publications - 14
Citations - 352
Ricardo Vaz Breda is an academic researcher from Pontifícia Universidade Católica do Rio Grande do Sul. The author has contributed to research in topics: Eosinophil peroxidase & Hippocampal formation. The author has an hindex of 8, co-authored 14 publications receiving 269 citations. Previous affiliations of Ricardo Vaz Breda include Allen Institute for Brain Science.
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Journal ArticleDOI
AM251, a selective antagonist of the CB1 receptor, inhibits the induction of long-term potentiation and induces retrograde amnesia in rats.
Lucas de Oliveira Alvares,Bruna Pasqualini Genro,Ricardo Vaz Breda,Michele Franzen Pedroso,Michele Franzen Pedroso,Jaderson Costa da Costa,Jaderson Costa da Costa,Jorge Alberto Quillfeldt +7 more
TL;DR: The role of the selective CB1 antagonist AM251 upon LTP induction in a hippocampal slice preparation is studied, and the effect of its intrahippocampal administration in the step-down inhibitory avoidance (IA) and the open field habituation tasks (OF).
Journal ArticleDOI
Mesenchymal stem cells improves survival in LPS‐induced acute lung injury acting through inhibition of NETs formation
Leonardo Pedrazza,Aline Andrea da Cunha,Carolina Luft,Nailê Karine Nunes,Felipe Schimitz,Rodrigo Benedetti Gassen,Ricardo Vaz Breda,Márcio Vinícius Fagundes Donadio,Angela T. S. Wyse,Paulo Márcio Pitrez,Jose Luis Rosa,Jarbas Rodrigues de Oliveira +11 more
TL;DR: For the first time, it is described that MSCs can reduce the formation of NETs and an experimental model of ALI, and this finding is directly related to these cells modulate the inflammatory response and oxidative damage in the course of the pathology.
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Autophagy induces eosinophil extracellular traps formation and allergic airway inflammation in a murine asthma model.
Josiane Silva Silveira,Géssica Luana Antunes,Daniela Benvenutti Kaiber,Mariana Severo da Costa,Fernanda Silva Ferreira,Eduardo Peil Marques,Felipe Schmitz,Rodrigo Benedetti Gassen,Ricardo Vaz Breda,Angela T. S. Wyse,Renato T. Stein,Paulo Márcio Pitrez,Aline Andrea da Cunha +12 more
TL;DR: Investigation of autophagy involvement in EETs release and in lung inflammation in an experimental asthma model shows that 3‐MA treatment can be an interesting alternative for reducing lung inflammation, oxidative stress, mitochondrial damage, and E ETs formation in asthma.
Journal ArticleDOI
Reactive oxygen species are involved in eosinophil extracellular traps release and in airway inflammation in asthma.
Josiane Silva Silveira,Géssica Luana Antunes,Daniela Benvenutti Kaiber,Mariana Severo da Costa,Eduardo Peil Marques,Fernanda Silva Ferreira,Rodrigo Benedetti Gassen,Ricardo Vaz Breda,Angela T. S. Wyse,Paulo Márcio Pitrez,Aline Andrea da Cunha +10 more
TL;DR: This is the first study to show that ROS are needed for EETs formation in asthma, and NAC and DPI treatments can be an interesting alternative for reducing airway inflammation, mitochondrial damage, and E ETs release in asthma.
Journal ArticleDOI
Altered distribution of striatal activity-dependent synaptic plasticity in the 3-nitropropionic acid model of Huntington's disease.
André Dalbem,Carine Volkweis Silveira,Michele Franzen Pedroso,Ricardo Vaz Breda,Cristiane von Werne Baes,Ana Paula Bartmann,Jaderson Costa da Costa +6 more
TL;DR: The results reinforce the idea that both forms of activity-dependent synaptic plasticity can be recorded at the dorsolateral region of striatum by the same stimulating protocol in control rats and suggest that 3-NP-induced striatal lesions may be associated with suppression of LTD expression in the sensorimotor striatum.