R
Richard E. Harlan
Researcher at Tulane University
Publications - 67
Citations - 3818
Richard E. Harlan is an academic researcher from Tulane University. The author has contributed to research in topics: Striatum & Nucleus accumbens. The author has an hindex of 28, co-authored 67 publications receiving 3734 citations. Previous affiliations of Richard E. Harlan include University Medical Center New Orleans & Roche Institute of Molecular Biology.
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Journal ArticleDOI
The accumbens: beyond the core-shell dichotomy.
Lennart Heimer,George F. Alheid,de Olmos Js,Henk J. Groenewegen,Suzanne N. Haber,Richard E. Harlan,Daniel S. Zahm +6 more
TL;DR: The shell, which is an especially diversified part of the accumbens, is the subject of special attention because of its close relation to the extended amygdala and distinctive response to antipsychotic and psychoactive drugs.
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Charting of Type II glucocorticoid receptor-like immunoreactivity in the rat central nervous system
R.S. Ahima,Richard E. Harlan +1 more
TL;DR: The rat brain and spinal cord has been mapped for Type II glucocorticoid receptor-like immunoreactivity in neurons and glia, using a monoclonal antibody, BUGR2, which recognizes an epitope close to the DNA-binding domain of the rat Type II receptor.
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Substantia innominata: a notion which impedes clinical–anatomical correlations in neuropsychiatric disorders
TL;DR: The accumbens shell and extended amygdala form an extensive forebrain continuum, which establishes specific neuronal circuits with the medial prefrontal-orbitofrontal cortex and medial temporal lobe, and is particularly characterized by a prominent system of long intrinsic association fibers.
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Type I corticosteroid receptor-like immunoreactivity in the rat CNS: distribution and regulation by corticosteroids.
TL;DR: This work has used two antisera against peptide sequences derived from the cDNA of the human Type I corticosteroid receptor to map the regional distribution and cortiosteroid regulation of the intracellular location of Type I Corticosteroids receptor‐like immunoreactivity (Type I‐ir) in the rat CNS.
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Drugs of abuse and immediate-early genes in the forebrain.
TL;DR: A review of the literature reporting activation of immediate-early gene expression in the forebrain, in response to administration of drugs of abuse, finds common neuropharmacological mechanisms involved in dopaminergic and glutamatergic systems.