R
Richard J. Robbins
Researcher at Cornell University
Publications - 48
Citations - 3872
Richard J. Robbins is an academic researcher from Cornell University. The author has contributed to research in topics: Thyroid cancer & Transplantation. The author has an hindex of 26, co-authored 48 publications receiving 3782 citations. Previous affiliations of Richard J. Robbins include Memorial Sloan Kettering Cancer Center & Yale University.
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Interictal Spikes and Hippocampal Somatostatin Levels in Temporal Lobe Epilepsy
TL;DR: A role of the EC in generation, regulation, or expression of interictal paroxysmal electrical activity in temporal lobe epilepsy (TLE), for which somatostatin may be a marker is suggested.
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Hypopituitarism associated with a hypothalamic CMV infection in a patient with AIDS.
William M. Sullivan,Grant G. Kelley,Patrick G. O'Connor,Philip S. Dickey,Jung H. Kim,Richard J. Robbins,Gerald I. Shulman +6 more
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Tyrosine hydroxylase immunoreactivity and monoamine and metabolite levels in cryopreserved human fetal ventral mesencephalon.
TL;DR: The results suggest that cryopreservation and subsequent thawing does not have a measurable adverse effect on DA biosynthesis in the human fetal mesencephalon.
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Growth promoting effects of IGF-I on fetal hypothalamic cell lines under serum-free culture conditions
TL;DR: Investigation of the effects of three insulin‐like peptides on thein vitro growth of fetal hypothalamic cell lines concluded that IGF‐I is a potent growth factor for virally transformed cell lines derived from the rat fetal hypothalamus, suggesting that IGF-I may constitute a mitogenic signal for hypothalamic cells during neurogenesis.
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Neural transplantation for neurodegenerative diseases: past, present, and future.
D. Eugene Redmond,D. Eugene Redmond,Robert H. Roth,Robert H. Roth,Dennis D. Spencer,Frederick Naftolin,Csaba Leranth,Richard J. Robbins,Kenneth Marek,John D. Elsworth,John D. Elsworth,Kimberlee J. Sass,Jane R. Taylor,Jane R. Taylor,John R. Sladek,John R. Sladek +15 more
TL;DR: If transplantation of brain cells can be accomplished successfully in humans, as it has been in animals, then replacement of a small population of dopamine‐producing cells in Parkinson's disease should have important functional effects and possibly reverse the course and symptoms of the disease.