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Robert E. Evans

Researcher at Fisheries and Oceans Canada

Publications -  41
Citations -  3746

Robert E. Evans is an academic researcher from Fisheries and Oceans Canada. The author has contributed to research in topics: Trout & Rainbow trout. The author has an hindex of 25, co-authored 41 publications receiving 3503 citations. Previous affiliations of Robert E. Evans include Government of Canada.

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Collapse of a fish population after exposure to a synthetic estrogen

TL;DR: It is shown that chronic exposure of fathead minnow (Pimephales promelas) to low concentrations of the potent 17α-ethynylestradiol led to feminization of males through the production of vitellogenin mRNA and protein, impacts on gonadal development as evidenced by intersex in males and altered oogenesis in females, and, ultimately, a near extinction of this species from the lake.
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Metabolism of selenomethionine by rainbow trout (Oncorhynchus mykiss) embryos can generate oxidative stress.

TL;DR: Methylselenol redox cycles in the presence of glutathione producing superoxide and likely accounts for oxidative lesions present in fish and birds environmentally exposed to excessive loads of selenomethionine.
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Toxicology of dietary nickel in lake whitefish (Coregonus clupeaformis)

TL;DR: To evaluate exposure of natural populations of fish to Ni, analysis of Ni in kidney and liver and assessment of renal and hepatic histopathology are recommended for use in field bio-monitoring programs.
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Developmental effects of bioaccumulated selenium in eggs and larvae of two salmonid species.

TL;DR: A significant relationship was observed for rainbow trout between the amount of Se in eggs and the incidence of developmental abnormalities, specifically craniofacial defects, skeletal deformities, and edema, similar to those described for centrarchids inhabiting a seleniferous warm-water lake.
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The characteristics of the electro-olfactogram (EOG): its loss and recovery following olfactory nerve section in rainbow trout (Salmo gairdneri).

TL;DR: The EOG responses evoked by HCl in sensory and non-sensory epithelia were indistinguishable from each other, indicating that receptor neurons are not likely to be their primary origin.