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Robert H. Bartlett

Researcher at University of Michigan

Publications -  569
Citations -  28295

Robert H. Bartlett is an academic researcher from University of Michigan. The author has contributed to research in topics: Extracorporeal membrane oxygenation & Extracorporeal. The author has an hindex of 81, co-authored 550 publications receiving 25619 citations. Previous affiliations of Robert H. Bartlett include University of California, Irvine & NewYork–Presbyterian Hospital.

Papers
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Increasing inspiratory time exacerbates ventilator-induced lung injury during high-pressure/high-volume mechanical ventilation.

TL;DR: Increasing inspiratory time during high-pressure/high-volume mechanical ventilation is associated with an increase in variables of lung injury and final static lung compliance and Pao2/Fio2 ratio decreased and wet/dry lung weight and dry lung/body weight decreased.
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A prototype of a liquid ventilator using a novel hollow-fiber oxygenator in a rabbit model.

TL;DR: The ability of this novel single-piston, nonporous hollow silicone fiber oxygenator to adequately support gas exchange, allowing successful performance of total liquid ventilation, is demonstrated.
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Extracorporeal life support after heart or lung transplantation

TL;DR: Ten patients after heart, lung, or heart-lung transplantation were used and survival was associated with younger age, shorter duration of ECLS, and longer interval from operation to initiation of ECRS but not to reason for initiating ECLs.
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Percutaneous access for venovenous extracorporeal life support in neonates

TL;DR: In the authors' early experience, percutaneous access using a 12F double lumen venovenous cannula may be efficiently performed especially in patients >3.0 kg in weight and has the potential for simplifying and reducing the cost of the ECLS technique.
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The Pulmonary and Systemic Distribution and Elimination of Perflubron From Adult Patients Treated With Partial Liquid Ventilation

TL;DR: Perflubron is eliminated from the lungs progressively at a maximum rate of 9.4 +/- 3.0 mL/h by evaporative loss from the airways and is retained in greater amounts in the dependent lung regions when compared to the nondependent lung regions.