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Robert J. Siegel

Researcher at Cedars-Sinai Medical Center

Publications -  506
Citations -  25581

Robert J. Siegel is an academic researcher from Cedars-Sinai Medical Center. The author has contributed to research in topics: Mitral regurgitation & Mitral valve. The author has an hindex of 75, co-authored 463 publications receiving 22687 citations. Previous affiliations of Robert J. Siegel include Northern General Hospital & University of California, Los Angeles.

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Echocardiographic detection of kaposi's sarcoma causing cardiac tamponade in a patient with acquired immunodeficiency syndrome

TL;DR: This is the first case of cardiac Kaposi's sarcoma detected premortem by echocardiography, diagnosed in a 32‐year‐old male with AIDS and systemic Kaposi' sarcoma.
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Left atrial pressure is associated with iatrogenic atrial septal defect after mitral valve clip.

TL;DR: Elevated LA pressure after MV clip was associated with persistent iASD and did not significantly differ from patients without 12M-iASD in terms of right heart enlargement, estimated systolic pulmonary artery pressure, New York Heart Association functional class and brain natriuretic peptide at 12 months.
Patent

Device for securing heart valve leaflets

TL;DR: In this article, a fixation device for securing together leaflets of a heart valve is provided, which is composed of two plates that are disposed on either side of the tricuspid valve.
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Echo-Doppler Hemodynamics An Important Management Tool for Today’s Heart Failure Care

TL;DR: A 52-year-old man with dilated cardiomyopathy, recently discharged from a heart failure (HF) admission, presented to the office with weakness and shortness of breath, and it was not clear whether his symptoms were related to progressive HF with worsening cardiorenal syndrome, or conversely, to relative hypovolemia.
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Predictors of reduction in mitral regurgitation in patients undergoing cardiac resynchronisation treatment

TL;DR: In patients with cardiomyopathy and significant MR, the presence of delayed longitudinal strain in the mid inferior LV segment along with preserved negative systolic strain inThe basal and mid posterior segments predicts substantial reduction in MR post-CRT.