Ryouhei Morita

TL;DR: The data collectively suggest that the identified SDR protein NYC1 plays essential roles in the regulation of LHCII and thylakoid membrane degradation during senescence.

TL;DR: Analysis of the nyc1 mutant, which shows the stay-green phenotype, and the nol mutant in rice, suggest that NOL and NYC1 are co-localized in the thylakoid membrane and act in the form of a complex as a chlorophyll b reductase in rice.

TL;DR: Functional analysis of sgr mutants in pea and rice suggests that SGR might be involved in activation of the Chl-degrading pathway during leaf senescence through translational or posttranslational regulation of ChL- degrading enzymes.

TL;DR: A new stay-green mutant in rice, nyc3, which encodes a plastid-localizing alpha/beta hydrolase-fold family protein with an esterase/lipase motif and the possible function of NYC3 in the regulation of chlorophyll degradation is discussed.

TL;DR: Reverse genetics applications may be possible for gamma irradiation-induced deletions in rice by mismatch cleavage analysis used in Targeting Induced Local Lesions IN Genomes (TILLING) to detect small deletions and base substitutions or by using array comparative genomic hybridization (aCGH), to detect large deletions.