S
S.J. Wysocki
Researcher at University of Western Australia
Publications - 10
Citations - 298
S.J. Wysocki is an academic researcher from University of Western Australia. The author has contributed to research in topics: Cancer & Gene expression. The author has an hindex of 7, co-authored 10 publications receiving 292 citations.
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Journal ArticleDOI
Overexpression of p53 protein is an independent prognostic indicator in human endometrial carcinoma
TL;DR: Immunohistochemical assessment of p53 overexpression in the nucleus and cytoplasm could provide useful prognostic information for the management of patients with endometrial cancer.
Journal ArticleDOI
Influence of Thyroid Hormones on Enzyme Activities of Myelinating Rat Central‐Nervous Tissues
S.J. Wysocki,Wolfe Segal +1 more
TL;DR: There is a critical period during the first 8 days of postnatal life when thyroid hormone levels must be adequate in order that spinal cord activity of this enzyme develops normally, it is concluded.
Journal ArticleDOI
Monocyte chemoattractant protein-1 gene expression in injured pig artery coincides with early appearance of infiltrating monocyte/macrophages.
S.J. Wysocki,Ming H. Zheng,Anne Smith,M.D. Lamawansa,Barry Iacopetta,Terry Robertson,John Papadimitriou,Anthony K. House,Paul Norman +8 more
TL;DR: The results are consistent with invading monocyte/macrophages having a major input into the production of this chemokine in the arterial wall following injury and suggestive that continued MCP‐1 expression is required for functions other than chemoattraction.
Journal Article
Detection of pS2 Messenger RNA in gynecological cancers
TL;DR: There was a poor correlation between estrogen receptor and pS2 mRNA in ovarian carcinomas, and the expression of the pS1 gene was analyzed in 111 gynecological carcinomas.
Journal ArticleDOI
The p53 tumor-suppressor gene is overexpressed but not mutated in human atherosclerotic tissue.
TL;DR: The results do not support the view that genetic alterations similar to those occurring in cancer contribute to the abnormal proliferation of SMC, although it is cautioned that only two cancer-related genes were studied.