S
Samantha L. Free
Researcher at University College London
Publications - 72
Citations - 5857
Samantha L. Free is an academic researcher from University College London. The author has contributed to research in topics: Epilepsy & Hippocampal sclerosis. The author has an hindex of 36, co-authored 72 publications receiving 5551 citations. Previous affiliations of Samantha L. Free include UCL Institute of Neurology.
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Journal ArticleDOI
Three-dimensional maximum probability atlas of the human brain, with particular reference to the temporal lobe
Alexander Hammers,Richard Allom,Matthias J. Koepp,Samantha L. Free,Ralph Myers,Louis Lemieux,Tejal N. Mitchell,David J. Brooks,John S. Duncan +8 more
TL;DR: This work has expanded and refined existing algorithms for the subdivision of MRI datasets into anatomical structures, and used the normalized superimposed atlases to create a maximum probability map in stereotaxic space, which retains quantitative information regarding inter‐subject variability.
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Abnormal cerebral structure in juvenile myoclonic epilepsy demonstrated with voxel-based analysis of MRI
TL;DR: Findings indicate a structural cerebral abnormality in juvenile myoclonic epilepsy, with involvement of mesiofrontal cortical structures.
Journal Article
Methods for normalization of hippocampal volumes measured with MR.
TL;DR: In this article, the use of six cerebral measures as correlates for hippocampal volumes was investigated, and corrected absolute hippocampal volume was determined for 32 control subjects and 23 patients with temporal lobe epilepsy.
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Surgical outcome in patients with epilepsy and dual pathology
Li Min Li,Fernando Cendes,Frederick Andermann,Craig Watson,David R. Fish,Mark J. Cook,François Dubeau,John S. Duncan,Simon Shorvon,Samuel F. Berkovic,Samantha L. Free,André Olivier,William Harkness,Douglas L. Arnold +13 more
TL;DR: The findings indicate that in patients with dual pathology removal of both the lesion and the atrophic hippocampus is the best surgical approach and should be considered whenever possible.
Journal ArticleDOI
Frequency and characteristics of dual pathology in patients with lesional epilepsy.
Fernando Cendes,Mark J. Cook,Craig Watson,F. Andermann,David R. Fish,Simon Shorvon,P. S. Bergin,Samantha L. Free,François Dubeau,Douglas L. Arnold +9 more
TL;DR: A common pathogenic mechanism during pre- or perinatal development may explain the occurrence of concomitant mesial temporal sclerosis and other structural lesions because of either associated developmental abnormalities or a predisposition to prolonged febrile convulsions.